Results 211 to 220 of about 75,185 (247)
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1984
The restriction patterns of genomes of twelve different field isolates of PrV were analyzed. All were found to differ. In most cases, the basis of the differences in restriction patterns was found to lie in the acquisition or deletion of sequences which appear to be nonessential to the productive infection of RK cells. Mutagenesis of a laboratory virus
Tamar Ben-Porat +5 more
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The restriction patterns of genomes of twelve different field isolates of PrV were analyzed. All were found to differ. In most cases, the basis of the differences in restriction patterns was found to lie in the acquisition or deletion of sequences which appear to be nonessential to the productive infection of RK cells. Mutagenesis of a laboratory virus
Tamar Ben-Porat +5 more
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Latency Sites and Reactivation of Duck Enteritis Virus
Avian Diseases, 2002Duck virus enteritis (DVE) is a contagious disease caused by herpesvirus in waterfowl populations. Recovered birds become carriers and shed the virus periodically. Reactivation of latent duck enteritis virus (DEV) has been implicated in outbreaks of DVE in domestic and migrating waterfowl populations.
Samia, Shawky, Karel A, Schat
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Latency and reactivation of infectious laryngotracheitis vaccine virus
Archives of Virology, 1991Latency and reactivation of a commercial infectious laryngotracheitis virus vaccine were demonstrated in live chickens. Virus was re-isolated at intervals between seven and fourteen weeks post-vaccination and this may be of epizootiological significance.
C S, Hughes +6 more
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Pseudorabies virus latency and reactivation in vaccinated swine
American Journal of Veterinary Research, 1990SUMMARY Latency and reactivation of pseudorabies virus in swine was studied. Thirty-one pigs were assigned to 5 groups and were given 1 of 4 vaccines; 10 remained unvaccinated controls. All pigs were then challenge exposed with a sublethal dose of virulent pseudorabies virus.
M A, Schoenbaum, G W, Beran, D P, Murphy
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Reactivation of Epstein-Barr virus from latency
Reviews in Medical Virology, 2005The general problem in cancer treatment centres on finding agents that specifically affect cancer cells without damaging normal cells. The differences between cancer cells and normal cells are usually very subtle but about 15% of all human cancers involve a virus infection, for example the Epstein-Barr virus associated cancers.
Wolfgang, Amon, Paul J, Farrell
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Neuronal Control of Herpes Simplex Virus Latency
Virology, 1993Herpes simplex virus (HSV) is a common neurotropic virus, and latent infection of sensory ganglion neurons readily occurs in humans and in experimentally infected animals. During HSV latency, infectious virus and viral antigen are not detected, and HSV transcription is limited to specific RNA termed latency-associated transcript (LAT).
R B, Tenser, W A, Edris, K A, Hay
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A mouse model for varicella-zoster virus latency
Microbial Pathogenesis, 1993Following primary infection with varicella-zoster virus (VZV), the virus establishes a latent infection in humans. The molecular pathogenesis of VZV latency is not well understood, mainly due to the lack of an adequate animal model. We report here that we have developed a mouse model for VZV infection that involves corneal inoculation of mice. Although
Z, Wroblewska +6 more
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The Molecular Basis for Human Immunodeficiency Virus Latency
Annual Review of Virology, 2017Although potent combination antiretroviral therapy can effectively block viral replication in the host, human immunodeficiency virus (HIV) persists due to the existence of latent but replication-competent proviruses residing primarily in a very small population of resting memory CD4+ T cells.
Uri, Mbonye, Jonathan, Karn
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Principles of Homeostasis in Governing Virus Activation and Latency
Immunologic Research, 2000The goal of our work is to understand, from the molecular to the organismal level, the principles that drive and sustain lifelong infection by viruses. These infectious agents live in a dynamic equilibrium (homeostasis) with their hosts in which both immune and nonimmune pathways contribute to viral homeostasis.
P, Ghazal +4 more
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VARICELLA-ZOSTER VIRUS LATENCY
Annual Review of Microbiology, 1991K D, Croen, S E, Straus
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