Results 61 to 70 of about 1,691,019 (337)

Weak solutions for some compressible multicomponent fluid models

open access: yes, 2018
The principle purpose of this work is to investigate a "viscous" version of a "simple" but still realistic bi-fluid model described in [Bresch, Desjardin, Ghidaglia, Grenier, Hillairet] whose "non-viscous" version is derived from physical considerations ...
Novotny, Antonin, Pokorny, Milan
core   +1 more source

Substrate specificity of Burkholderia pseudomallei multidrug transporters is influenced by the hydrophilic patch in the substrate‐binding pocket

open access: yesFEBS Letters, EarlyView.
Multidrug transporters BpeB and BpeF from the Gram‐negative pathogen Burkholderia pseudomallei have a hydrophilic patch in their substrate‐binding pocket. Drug susceptibility tests and growth curve analyses using an Escherichia coli recombinant expression system revealed that the hydrophilic patches of BpeB and BpeF are involved in the substrate ...
Ui Okada, Satoshi Murakami
wiley   +1 more source

Global well-posedness for the dynamical Q-tensor model of liquid crystals

open access: yes, 2015
In this paper, we consider a complex fluid modeling nematic liquid crystal flows, which is described by a system coupling Navier-Stokes equations with a parabolic Q-tensor system.
Ding, Shijin, Huang, Jinrui
core   +1 more source

Weak Solutions of the Porous Medium Equation [PDF]

open access: yesTransactions of the American Mathematical Society, 1993
We show that if u ≥ 0 u \geq 0 , u ∈ L loc m ( Ω ) u \in L_{{\text {loc}}}^m(\Omega ) , Ω ⊂ R
Dahlberg, Björn E. J., Kenig, Carlos E.
openaire   +2 more sources

β‐TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1

open access: yesMolecular Oncology, EarlyView.
Low levels of β‐TrCP (Panel A) allow the accumulation of BRCA1 and CtIP, which facilitate the repair of cisplatin‐induced DNA damage via homologous recombination (HR) and promote tumor cell survival. In contrast, high β‐TrCP expression (Panel B) leads to BRCA1 and CtIP degradation, impairing HR repair, resulting in persistent DNA damage and apoptosis ...
Rocío Jiménez‐Guerrero   +8 more
wiley   +1 more source

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

open access: yesMolecular Oncology, EarlyView.
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian   +10 more
wiley   +1 more source

Weak solutions for quasilinear degenerate parabolic systems

open access: yesElectronic Journal of Differential Equations, 2006
This paper concerns the initial Dirichlet boundary-value problem for a class of quasilinear degenerate parabolic systems. Due to the degeneracies, the problem does not have classical solutions in general.
Zheng'an Yao, Wenshu Zhou
doaj  

Mean field type control with congestion [PDF]

open access: yes, 2015
We analyze some systems of partial differential equations arising in the theory of mean field type control with congestion effects. We look for weak solutions.
Achdou, Yves, Lauriere, Mathieu
core   +2 more sources

Weak-strong uniqueness of solutions to entropy-dissipating reaction-diffusion equations

open access: yes, 2017
We establish a weak-strong uniqueness principle for solutions to entropy-dissipating reaction-diffusion equations: As long as a strong solution to the reaction-diffusion equation exists, any weak solution and even any renormalized solution must coincide ...
Alexandre   +37 more
core   +1 more source

Emerging role of ARHGAP29 in melanoma cell phenotype switching

open access: yesMolecular Oncology, EarlyView.
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster   +3 more
wiley   +1 more source

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