Results 71 to 80 of about 67,575 (268)

Poly-N-Acetylglucosamine Expression by Wild-Type Yersinia pestis Is Maximal at Mammalian, Not Flea, Temperatures [PDF]

, 2012
Numerous bacteria, including Yersinia pestis, express the poly-N-acetylglucosamine (PNAG) surface carbohydrate, a major component of biofilms often associated with a specific appearance of colonies on Congo red agar.
Cywes-Bentley, Colette, Pier, Gerald B., Yoong, Pauline   +2 more
core   +1 more source

The Origin of Fleas and the Genesis of Plague [PDF]

, 2015
Human history has been riddled by diseases spread by flea vectors including the bubonic plague. Recently, Madagascar has documented more than 100 cases.
Gillen, Alan L.
core   +1 more source

'Add, stir and reduce': Yersinia spp. as model bacteria for pathogen evolution [PDF]

, 2016
In the study of molecular microbiology and bacterial genetics, pathogenic species of the Yersinia genus have been pillars for research aimed at understanding how bacteria evolve into mammalian pathogens.
McNally, A, Reuter, S, Thomson, NR, Wren, BW   +3 more
core   +3 more sources

Phylogeny and Classification of Yersinia pestis Through the Lens of Strains From the Plague Foci of Commonwealth of Independent States

Frontiers in Microbiology, 2018
The established phylogeny of the etiological agent of plague, Yersinia pestis, is not perfect, as it does not take into account the strains from numerous natural foci of Commonwealth of Independent States (CIS).
V. Kutyrev, G. A. Eroshenko, V. Motin, N. Nosov, Ja. M. Krasnov, L. M. Kukleva, K. A. Nikiforov, Z. V. Al’khova, E. G. Oglodin, N. P. Guseva   +9 more
semanticscholar   +1 more source

The NLRP12 Inflammasome Recognizes Yersinia pestis [PDF]

Immunity, 2012
Yersinia pestis, the causative agent of plague, is able to suppress production of inflammatory cytokines IL-18 and IL-1β, which are generated through caspase-1-activating nucleotide-binding domain and leucine-rich repeat (NLR)-containing inflammasomes. Here, we sought to elucidate the role of NLRs and IL-18 during plague. Lack of IL-18 signaling led to
Vijay A. K. Rathinam, John Bertin, Joan Mecsas, Megan K. Proulx, George W. Reed, Yoichiro Iwakura, Jon D. Goguen, Qin Liu, Katherine A. Fitzgerald, Marie Hjelmseth Aune, Dan Weng, Gregory I. Vladimer, Sara W. Montminy Paquette, Joseph J. Burbage, Egil Lien, Egil Lien, Joseph E. Conlon, Sivapriya Kailasan Vanaja   +17 more
openaire   +5 more sources

Host Langerin (CD207) is a receptor for Yersinia pestis phagocytosis and promotes dissemination [PDF]

, 2015
Yersinia pestis is a Gram-negative bacterium that causes plague. After Y. pestis overcomes the skin barrier, it encounters antigen-presenting cells (APCs), such as Langerhans and dendritic cells.
Anyi Li, Chae G Park, Cheolho Cheong, Gottfried RS., Gregory V Plano, Honghui Ding, Hongping Huang, John D Klena, Kun Yang, Lianxu Xia, Lin Zhang, Lingyu Jiang, Mikael Skurnik, Pei Zhang, Sara S Bartra, Shaogang Wang, Shusheng Zhang, Silvia Bulgheresi, Tie Chen, Yingxia He, Yiping Wu   +20 more
core   +1 more source

Integrative approach using Yersinia pestis genomes to revisit the historical landscape of plague during the Medieval Period

Proceedings of the National Academy of Sciences of the United States of America, 2018
Significance While our knowledge of modern plague reservoirs and their hosts is extensive, we have little to no knowledge about the origin of the Medieval plague pandemics or the routes of transmission involved in their spread. Prior genomic data provide
A. Namouchi, Meriam Guellil, Oliver Kersten, S. Hänsch, C. Ottoni, Boris V. Schmid, E. Pacciani, L. Quaglia, Marco Vermunt, E. L. Bauer, Michael Derrick, A. Ø. Jensen, S. Kacki, S. Cohn, N. Stenseth, B. Bramanti   +15 more
semanticscholar   +1 more source

Intraspecific Diversity ofYersinia pestis [PDF]

Clinical Microbiology Reviews, 2004
SUMMARYIncreased interest in the pathogenic potential ofYersinia pestishas emerged because of the potential threats from bioterrorism. Pathogenic potential is based on genetic factors present in a population of microbes, yet most studies evaluating the role of specific genes in virulence have used a limited number of strains. ForY.
Gerald B. Pier, Luther E. Lindler, Andrey P. Anisimov   +2 more
openaire   +4 more sources

Characteristics of an antioxidant activity of Yersinia pestis with different plasmid spectrum

Acta Biomedica Scientifica, 2016
Experimental data concerning complex study of antioxidant activity of Y. pestis with different plasmid spectrum (wild-type Yersinia pestis subsp. pestis, Yersinia pestis subsp.
O. V. Yuryeva, V. I. Dubrovina, K. M. Korytov, G. B. Mukhturgin, T. A. Ivanova, S. A. Vityazeva, E. G. Tokmakova, S. V. Balakhonov   +7 more
doaj   +1 more source

A High-Coverage Yersinia pestis Genome from a Sixth-Century Justinianic Plague Victim

Molecular biology and evolution, 2016
The Justinianic Plague, which started in the sixth century and lasted to the mid eighth century, is thought to be the first of three historically documented plague pandemics causing massive casualties.
M. Feldman, M. Harbeck, Marcel Keller, M. Spyrou, Andreas Rott, B. Trautmann, H. Scholz, B. Päffgen, J. Peters, M. McCormick, K. Bos, A. Herbig, J. Krause   +12 more
semanticscholar   +1 more source