Results 31 to 40 of about 290,887 (206)

Assessing chronic rhinosinusitis with nasal polyps severity by “Japanese epidemiological survey of refractory eosinophilic chronic rhinosinusitis” algorithm [PDF]

open access: yesExploration of Asthma & Allergy, 2023
Aim: Chronic rhinosinusitis with nasal polyps (CRSwNP) is a complex disease with different subtypes that affect patients’ quality of life. This study aim to evaluate the severity of CRSwNP and the risk of treatment resistance using the “Japanese ...
Sérgio Duarte Dortas Jr   +7 more
doaj   +1 more source

The lncRNA PVT1/miR-590-5p/FSTL1 axis modulates the proliferation and migration of airway smooth muscle cells in asthma

open access: yesAutoimmunity, 2021
Objective Asthma is a prevalent chronic inflammatory airway disease that is characterised by airway remodelling and airway hyperresponsiveness. Abnormal proliferation and migration of airway smooth muscle cells (ASMCs) contribute to airway remodelling in
Wen-Lan Wang   +5 more
doaj   +1 more source

Selection of a Navigation Strategy According to Agricultural Scenarios and Sensor Data Integrity

open access: yesAgronomy, 2023
In digital farming, the use of technology to increase agricultural production through automated tasks has recently integrated the development of AgBots for more reliable data collection using autonomous navigation.
Leonardo Bonacini   +9 more
doaj   +1 more source

MiR-590-3p affects the function of adipose-derived stem cells (ADSCs) on the survival of skin flaps by targeting VEGFA

open access: yesRegenerative Therapy, 2022
Introduction: Partial necrosis of skin flaps is still a substantial problem in plastic and reconstructive surgery. In this study, the role of miR-590-3p in adipose-derived stem cells (ADSCs) transplantation in improving the survival of skin flap in a ...
Kai Yang   +7 more
doaj   +1 more source

Digital economy policies and corporate technology spillovers: A data-driven perspective

open access: yesInternational Review of Economics & Finance
Efficient data aggregation and allocation are crucial for driving scientific and technological innovation, resource allocation efficiency, and technology spillovers.
Zhanyu Zhang   +3 more
doaj   +1 more source

MicroRNA-590-3p promotes cell proliferation and invasion by targeting inositol polyphosphate 4-phosphatase type II in human prostate cancer cells

open access: yesTumor Biology, 2017
Inositol polyphosphate 4-phosphatase type II emerges as a tumor suppressor in prostate cancer, and its loss of expression is associated with poor prognosis for prostate cancer.
Haiwen Chen, Qidong Luo, Hongliang Li
doaj   +1 more source

The miR-590 C57T SNP reduces levels of miR-590-5p and miR-590-3p, without affecting the levels of pri-miR-590 and pre-miR-590.

open access: yes, 2016
(A) Quantification of pri-miR-590 by qRT-PCR normalized by GAPDH. Mean ± SD (n = 3). HEK293T cells were transfected with the pri-miR-590-WT or pri-miR-590-SNP plasmids. The empty plasmid was used as negative control.
Suresh K. Kandasamy (2794198)   +15 more
core   +1 more source

Figure S2 from miR-590-3p Promotes Ovarian Cancer Growth and Metastasis via a Novel FOXA2–Versican Pathway

open access: yes, 2018
Generation of EOC cell lines stably over expressing mir-590 and the effect of mir-590 over expression on colony ...
Chun Peng (258176)   +11 more
core   +1 more source

Supplementary Material for: miR-590-3p Is a Novel MicroRNA in Myocarditis by Targeting Nuclear Factor Kappa-B in vivo

open access: yes, 2017
Objective: Nuclear factor kappa-B (NF-κB)-induced inflammation leads to myocarditis and heart dysfunction. How microRNAs (miRNAs) contribute to this process is poorly defined. The aim of this study was to investigate whether miRNAs regulate NF-κB-induced
Zhou J. (3931118)   +9 more
core   +1 more source

Figure S1 from miR-590-3p Promotes Ovarian Cancer Growth and Metastasis via a Novel FOXA2–Versican Pathway

open access: yes, 2018
miR-590-3p promotes cell proliferation, migration, and invasion in multiple EOC cell lines.
Chun Peng (258176)   +11 more
core   +1 more source

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