Results 211 to 220 of about 89,390 (263)
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Archives of Internal Medicine, 1977
This book is a logical and welcome successor to the author's previous monograph published in 1959. It combines a broad-brush as well as a detailed analysis of various points and controversies within the aldosterone literature. Two criticisms are offered.
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This book is a logical and welcome successor to the author's previous monograph published in 1959. It combines a broad-brush as well as a detailed analysis of various points and controversies within the aldosterone literature. Two criticisms are offered.
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Regulation of Aldosterone Secretion in Primary Aldosteronism
Hormone and Metabolic Research, 1975Plasma aldosterone, plasma renin activity and plasma cortisol were determined in patients with primary aldosteronism in response to posture and at short-time intervals overnight while the patient were supine. In the 5 patients with an aldosterone-producing adenoma postural changes in plasma aldosterone were paralleled by those in cortisol while plasma ...
H, Vetter, W, Vetter
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Aldosterone and aldosterone antagonism in systemic hypertension
Current Hypertension Reports, 2004Aldosterone mediates both water and electrolyte balance by acting on the renal mineralocorticoid receptors. Recent experimental studies have also documented the presence of these receptors in other body organs, including the brain, blood vessels, and heart, suggesting that aldosterone plays a larger role in normal physiologic function and in ...
William H, Frishman, Charles T, Stier
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Journal of Surgical Oncology, 2009
AbstractPrimary aldosteronism is the most common form of secondary hypertension. Case detection is based on an abnormal plasma aldosterone:plasma renin activity ratio and the diagnosis must be confirmed with an aldosterone suppression test. Subtype differentiation should be performed using adrenal venous sampling.
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AbstractPrimary aldosteronism is the most common form of secondary hypertension. Case detection is based on an abnormal plasma aldosterone:plasma renin activity ratio and the diagnosis must be confirmed with an aldosterone suppression test. Subtype differentiation should be performed using adrenal venous sampling.
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Trends in Cardiovascular Medicine, 2016
In 1955 Dr Jerome Conn first documented primary aldosteronism (PA). Since then, screening, diagnosis and treatment have developed, in the process both refining and complicating management. Currently, screening requires 4-6 weeks of lead-up, including major changes in antihypertensive therapy, followed by a blood draw for plasma aldosterone ...
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In 1955 Dr Jerome Conn first documented primary aldosteronism (PA). Since then, screening, diagnosis and treatment have developed, in the process both refining and complicating management. Currently, screening requires 4-6 weeks of lead-up, including major changes in antihypertensive therapy, followed by a blood draw for plasma aldosterone ...
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The regulation of aldosterone secretion in primary aldosteronism
The American Journal of Medicine, 1972Abstract Twenty patients with primary aldosteronism were studied under various conditions known to influence aldosterone secretion. All patients presented with the classic findings of hypertension, hypokalemia, suppressed plasma renin activity and nonsuppressible aldosterone secretion.
J P, Cain +4 more
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Suppression of Aldosterone by Cyproheptadine in Idiopathic Aldosteronism
New England Journal of Medicine, 1981To study the role of serotonin in regulating the release of aldosterone, we gave single, oral doses of cyproheptadine, an antiserotoninergic agent, to five normal volunteers with high aldosterone levels secondary to sodium deprivation and to 14 patients with aldosteronism (six with idiopathic aldosteronism due to bilateral adrenal hyperplasia and eight
M D, Gross +3 more
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Journal of Endocrinological Investigation, 1995
The basic clinical pathophysiology of primary aldosteronism (PAL) was described by Conn in terms of autonomous production of aldosterone, secondary suppression of renin and development of hypertension with hypokalaemic alkalosis. Conn recognised a normokalaemic form of the syndrome and suggested that it might masquerade as essential hypertension and be
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The basic clinical pathophysiology of primary aldosteronism (PAL) was described by Conn in terms of autonomous production of aldosterone, secondary suppression of renin and development of hypertension with hypokalaemic alkalosis. Conn recognised a normokalaemic form of the syndrome and suggested that it might masquerade as essential hypertension and be
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ALDOSTERONE METABOLITES AND POSSIBLE ALDOSTERONE PRECURSORS IN HYPERTENSION
Journal of Steroid Biochemistry, 1983The value of the urine tests: free aldosterone, aldosterone-18-glucuronide, tetrahydroaldosterone 18-hydroxycorticosterone and 18-hydroxydeoxycorticosterone in distinguishing primary aldosteronism from essential hypertension was studied in patients with typical and atypical primary aldosteronism and in patients with essential hypertension.
P, Vecsei +5 more
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The Journal of Clinical Endocrinology & Metabolism, 1955
I 1934 an amorphous fraction of adrenal extracts was recognized to have a particular^ high life-maintaining activity. Oddly enough, although knowledge of other adrenal hormones accumulated in profusion, it took two decades before a specific crystalline component of this fraction, aldosterone (electrocortin), was isolated and characterized.
R, GAUNT, A A, RENZI, J J, CHART
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I 1934 an amorphous fraction of adrenal extracts was recognized to have a particular^ high life-maintaining activity. Oddly enough, although knowledge of other adrenal hormones accumulated in profusion, it took two decades before a specific crystalline component of this fraction, aldosterone (electrocortin), was isolated and characterized.
R, GAUNT, A A, RENZI, J J, CHART
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