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Stress-induced alteration of small extracellular vesicles drives amyloid-beta sequestration and exacerbates Alzheimer's disease pathogenesis. [PDF]
Zehra S +13 more
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Type I interferon drives T cell responses to amyloid beta in the central nervous system. [PDF]
Michel JJ +13 more
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ESC Heart Failure, Volume 12, Issue 2, Page 727-729, April 2025.
Wojciech Kosmala +1 more
wiley +1 more source
Landscape of Apolipoprotein (APOE) in the Emirati Population and Recommendations for a Genotyping Approach for Amyloid-Beta Monoclonal Antibodies (Aβ-mAbs) in Alzheimer's Disease Patients. [PDF]
Mohammed W +16 more
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Alzheimer's Disease Risk Variants Interact with Amyloid-Beta to Modulate Monocyte Function. [PDF]
Chatila ZK, Bradshaw EM.
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Crocetin inhibits beta-amyloid fibrillization and stabilizes beta-amyloid oligomers
Biochemical and Biophysical Research Communications, 2011Aggregation of a peptide, beta-amyloid (Aβ), is a hallmark molecular process found in Alzheimer's disease (AD). During Aβ aggregation, oligomeric and fibrillar Aβ are formed, and these molecular self-assembly steps are implicated in generation of toxic effects in AD.
Joon Ho, Ahn +3 more
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2019
Alzheimer's disease (AD), exhibiting accumulation of amyloid beta (Aβ) peptide as a foremost protagonist, is one of the top five causes of deaths. It is a neurodegenerative disorder (ND) that causes a progressive decline in memory and cognitive abilities. It is characterized by deposition of Aβ plaques and neurofibrillary tangles (NFTs) in the neurons,
Abhinav Anand +3 more
openaire +1 more source
Alzheimer's disease (AD), exhibiting accumulation of amyloid beta (Aβ) peptide as a foremost protagonist, is one of the top five causes of deaths. It is a neurodegenerative disorder (ND) that causes a progressive decline in memory and cognitive abilities. It is characterized by deposition of Aβ plaques and neurofibrillary tangles (NFTs) in the neurons,
Abhinav Anand +3 more
openaire +1 more source

