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Research Anthology on Diagnosing and Treating Neurocognitive Disorders, 2019
Alzheimer's disease (AD), exhibiting accumulation of amyloid beta (Aβ) peptide as a foremost protagonist, is one of the top five causes of deaths. It is a neurodegenerative disorder (ND) that causes a progressive decline in memory and cognitive abilities. It is characterized by deposition of Aβ plaques and neurofibrillary tangles (NFTs) in the neurons,
Abhinav Anand +3 more
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Alzheimer's disease (AD), exhibiting accumulation of amyloid beta (Aβ) peptide as a foremost protagonist, is one of the top five causes of deaths. It is a neurodegenerative disorder (ND) that causes a progressive decline in memory and cognitive abilities. It is characterized by deposition of Aβ plaques and neurofibrillary tangles (NFTs) in the neurons,
Abhinav Anand +3 more
openaire +2 more sources
NeuroImage, 2019
Beta amyloid is a protein fragment snipped from the amyloid precursor protein (APP). Aggregation of these peptides into amyloid plaques is one of the hallmarks of Alzheimer's disease.
Nan-Jie Gong +2 more
exaly +2 more sources
Beta amyloid is a protein fragment snipped from the amyloid precursor protein (APP). Aggregation of these peptides into amyloid plaques is one of the hallmarks of Alzheimer's disease.
Nan-Jie Gong +2 more
exaly +2 more sources
Review on Alzheimer's disease: Inhibition of amyloid beta and tau tangle formation.
International Journal of Biological Macromolecules, 2020It is reported that approximately 40 million people are suffering from dementia, globally. Dementia is a group of symptoms that affect neurons and cause some mental disorders, such as losing memory.
Hossein Ashrafian +2 more
semanticscholar +1 more source
Crocetin inhibits beta-amyloid fibrillization and stabilizes beta-amyloid oligomers
Biochemical and Biophysical Research Communications, 2011Aggregation of a peptide, beta-amyloid (Aβ), is a hallmark molecular process found in Alzheimer's disease (AD). During Aβ aggregation, oligomeric and fibrillar Aβ are formed, and these molecular self-assembly steps are implicated in generation of toxic effects in AD.
Joon Ho, Ahn +3 more
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Clearance of Beta-Amyloid in the Brain
Current Medicinal Chemistry, 2014Intravascular substances invade extracellular spaces in the brain via endothelial cells in the sites without bloodbrain barrier (BBB) and move not only in the cerebrospinal fluid (CSF) but also in the interstitial fluid (ISF) of brain parenchyma adjacent to non-BBB sites.
Masaki, Ueno +4 more
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