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Hereditary angioedema type III (estrogen-dependent) report of three cases and literature review* [PDF]
Anais Brasileiros de Dermatologia, 2013 In this article, three cases of hereditary angioedema (HAE) type III (estrogen-dependent or with normal C1 inhibitor) are reported. The HAE was initially described in women of the same family in association with high-leveled estrogenic conditions such as
Amanda Rodrigues Miranda +5 more
doaj +1 more source
Optimum Use of Acute Treatments for Hereditary Angioedema: Evidence-Based Expert Consensus
Frontiers in Medicine, 2018 Acute treatment of hereditary angioedema due to C1 inhibitor deficiency has become available in the last 10 years and has greatly improved patients’ quality of life.
Hilary Longhurst
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Physician awareness and understanding of hereditary angioedema: A web‐based study in Japan
Journal of Cutaneous Immunology and Allergy, 2022 Objectives Hereditary angioedema (HAE) is a rare disease with acute attacks in the skin and mucosa throughout the body including life‐threatening laryngeal edema and abdominal attacks with severe pain.
Atsushi Fukunaga +6 more
doaj +1 more source
Frontiers in Immunology, 2021 It has been hypothesized that low levels of C1 esterase inhibitor (C1-INH), a key inhibitor of the complement pathway, may play a role in the occurrence of adverse events (AEs) associated with intravenous immunoglobulin (IVIG) therapy.
Isaac R. Melamed +3 more
doaj +1 more source
Revista Alergia México, 2016 Angioedema is defined as edema of the skin or mucosa, including the respiratory and the gastrointestinal mucosa, which is self-limiting, and in most cases is completely resolved in less than 72 hours. It occurs due to increased permeability of the mucosal and submucosal capillaries and postcapillary venules, with resulting plasma extravasation.
Holguín Gómez, Luisa +2 more
openaire +6 more sources
Allergy, Asthma & Clinical Immunology, 2010 Acquired angioedema (AAE) is characterized by acquired deficiency of C1 inhibitor (C1-INH), hyperactivation of the classical pathway of human complement and angioedema symptoms mediated by bradykinin released by inappropriate activation of the contact-kinin system.
M. Cicardi, A. Zanichelli
openaire +3 more sources
Acta Médica Portuguesa, 2019 Introduction: Hospitalizations due to angioedema are important especially in debilitating or life-threatening situations. The aim of this study was to evaluate the frequency and etiology of angioedema without urticaria in hospital admissions.
Joana Cosme +3 more
doaj +1 more source
Cureus, 2023 The authors present the case of a 22-year-old female who reported having a persistent sore throat. The patient had a history of recurring episodes of hereditary angioedema and arrived at the emergency department with her C1-esterase inhibitor. The epidemiology, clinical presentation, and treatment strategies are presented.
Banerjee, Anjali +4 more
openaire +2 more sources
Advanced Science, EarlyView.This study unveils an unrecognized pro‐inflammatory epitranscriptomic checkpoint in psoriasis. By installing m7G modifications on the 5′ UTR of Bdkrb1 mRNA, METTL1 enhances receptor stability to orchestrate keratinocyte‐driven neutrophil recruitment via p38 MAPK signaling.
Chang Zhang +10 more
wiley +1 more source
Anais Brasileiros de Dermatologia, 2012 Hereditary Angioedema is a dominantly inherited disease. Routine screening of autoantibodies (AAB) is not recommended for individuals with Hereditary Angioedema; however, prevalence of these antibodies in Hereditary Angioedema patients is not well ...
Sergio Duarte Dortas Junior +7 more
doaj +1 more source