Results 171 to 180 of about 74,493 (218)

Relapse of subacute anti-glomerular basement membrane nephritis following self-discontinuation of steroid therapy. [PDF]

CEN Case Rep
Takenaka Y, Hashiba T, Ogura Y, Ando S, Fujii A, Mise N.   +5 more
europepmc   +1 more source

The renal response to FGF23 shifts from phosphaturia toward inflammation in kidney disease. [PDF]

J Cell Commun Signal
Moor MB, Burmakin M, Levin A, Korkut GG, Brodin D, Wernerson A, Bruchfeld A, Bárány P, Witasp A, Patrakka J, Olauson H.   +10 more
europepmc   +1 more source

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Relationship between anti-GBM antibodies and kidney outcomes in patients with anti-GBM disease

Journal of Nephrology, 2022
Abstract Introduction Animal experiments have shown that anti-GBM antibodies play a pathogenic role in anti-GBM disease. However, the relationship between anti-GBM antibody levels and kidney outcomes in patients with anti-GBM disease is unclear.
Ying Jin
exaly   +3 more sources

Diagnosis and classification of Goodpasture's disease (anti-GBM)

Journal of Autoimmunity, 2014
Goodpasture's disease or anti-glomerular basement membrane disease (anti-GBM-disease) is included among immune complex small vessel vasculitides. The definition of anti-GBM disease is a vasculitis affecting glomerular capillaries, pulmonary capillaries, or both, with GBM deposition of anti-GBM autoantibodies.
Thomas Hellmark, Marten Segelmark
exaly   +5 more sources

Anti-GBM disease with a mild relapsing course and low levels of anti-GBM autoantibodies [PDF]

CKJ: Clinical Kidney Journal, 2012
Anti-glomerular basement membrane disease (anti-GBM) is usually characterized by rapidly progressive glomerulonephritis, and when autoantibody production has ceased, relapses are rare. Here, we report a 71-year-old women diagnosed at a stage of mild renal insufficiency.
Marten Segelmark, Jorgen Wieslander, Segelmark Marten   +2 more
exaly   +3 more sources

“Atypical” Anti-GBM Disease: Just Atypical, or Not Anti-GBM Disease at All?

American Journal of Kidney Diseases
Two large cohorts1,2 of patients with “atypical” anti–glomerular basement membrane (GBM) disease sharedheterogeneous clinical presentations and a single commoninclusioncriterion—linearIgGstainingalongthe GBM, foundonbiopsy. Indeed,4patientsevendemonstratedanti-GBMantibodies, deemedrelevantforthediagnosisof“typical”anti-GBMdis ease ...
Vanja Ivković, Andreas Kronbichler, Tiffany N. Caza   +2 more
openaire   +2 more sources

Atypical anti-GBM disease in pregnancy

BMJ Case Reports
A woman in her 20s presented with nephrotic syndrome and hyperemesis in early pregnancy. Pertinent initial investigations revealed a severe acute kidney injury, a serum albumin of 19 g/L, a random protein creatinine ratio of 800 g/mol and microscopic haematuria.
Shaun Chandler, Dharmenaan Palamuthusingam   +1 more
openaire   +4 more sources

Anti-GBM disease and ANCA during dengue infection

Clinical Nephrology, 2015
Anti-glomerular basement membrane (GBM) disease is a severe inflammatory renal disorder due to pathogenic autoantibodies directed mainly against the α3 chain of type IV collagen. In ~1/4 of patients with anti-GBM disease, antineutrophil cytoplasmic antibodies (ANCA) predominantly with myeloperoxidase (MPO) specificity can be detected.
Karlo J, Lizarraga, Jorge A, Florindez, Pirouz, Daftarian, David M, Andrews, Luis M, Ortega, Jair Munoz, Mendoza, Gabriel N, Contreras, Ali, Nayer   +7 more
openaire   +2 more sources

Lambda-Light-Chain-Mediated Anti-GBM Disease

Nephron, 2008
A 77-year-old man presented with macroscopic haematuria and oligo-anuric renal failure. A renal biopsy showed moderate linear glomerular basement membrane (GBM) staining with λ light chains alone, but both circulating IgG and λ light chain activity were present. There was no evidence of a serum or urinary paraprotein and a bone marrow biopsy was normal.
J A, Savige, S P, Yeung, A R, Bierre, P, Kincaid-Smith   +3 more
openaire   +2 more sources