Antibiotic‐mediated immune modulation in periodontitis
Abstract Periodontitis is a chronic inflammatory disease affecting the supporting structures of the teeth. Although initiated by dysbiotic microbial communities, its progression is largely driven by the host's uncontrolled inflammatory response. While antibiotics have conventionally been employed in periodontitis therapy for their antimicrobial ...
Lina J. Suárez +6 more
wiley +1 more source
Calcium signaling: an emerging player in plant antiviral defense. [PDF]
Zvereva AS, Klingenbrunner M, Teige M.
europepmc +1 more source
Role of RIPK1/RIPK3/MLKL signalling pathway in sepsis‐associated acute kidney injury
Abstract Sepsis‐associated acute kidney injury (SA‐AKI) is a common clinical syndrome in critically ill patients, and its high mortality rate is closely related to complex pathological mechanisms. Existing studies have shown that the pathophysiological process of SA‐AKI involves complex multi‐mechanism interactions, including an uncontrolled systemic ...
Huijun Yin, Jingyi Wang, Huirong Han
wiley +1 more source
Tissue-specific features of innate lymphoid cells in antiviral defense. [PDF]
Piersma SJ.
europepmc +1 more source
An OLD protein teaches us new tricks: prokaryotic antiviral defense. [PDF]
Ednacot EMQ, Morehouse BR.
europepmc +1 more source
Functional characterization of a novel protein-coding circular RNA, circRNA_1193, from the <i>mAAP</i> gene in silkworm and its role in antiviral defense against BmCPV. [PDF]
Li S +9 more
europepmc +1 more source
Unraveling Novel Strategies: Targeting Miz1 for Degradation to Enhance Antiviral Defense against Influenza A Virus. [PDF]
Xia B, Zhao J.
europepmc +1 more source
The evolutionary trajectories of specialized metabolites towards antiviral defense system in plants. [PDF]
Ahmad N, Xu Y, Zang F, Li D, Liu Z.
europepmc +1 more source
RNA helicase SKIV2L limits antiviral defense and autoinflammation elicited by the OAS-RNase L pathway. [PDF]
Yang K +4 more
europepmc +1 more source
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Trends in Immunology, 2022Effector-triggered immunity (ETI) is a common defense strategy used by mammalian host cells that is engaged upon detection of the enzymatic activities of pathogen-encoded proteins or the effects of their expression on cellular homeostasis. However, in contrast to the effector-triggered responses engaged upon bacterial infection, much less is understood
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