Results 1 to 10 of about 360,361 (166)

Atherosclerosis [PDF]

open access: yesNature Reviews Disease Primers, 2019
Atherosclerosis, the formation of fibrofatty lesions in the artery wall, causes much morbidity and mortality worldwide, including most myocardial infarctions and many strokes, as well as disabling peripheral artery disease. Development of atherosclerotic lesions probably requires low-density lipoprotein, a particle that carries cholesterol through the ...
Peter Libby   +2 more
exaly   +5 more sources

Atherosclerosis [PDF]

open access: yesAnnals of the Rheumatic Diseases, 2002
Is atherosclerosis a cellular or humoral mediated autoimmune disease?
Y, Sherer, Y, Shoenfeld
openaire   +2 more sources

Partial duplication of the EGF precursor homology domain of the LDL-receptor protein causing familial hypercholesterolemia (FH-Salerno).

open access: yesJournal of Lipid Research, 1994
A novel mutation of low density lipoprotein (LDL)-receptor gene was found in an Italian family hypercholesterolemia (FH) patient during a screening of 300 FH patients.
S Bertolini   +9 more
doaj   +1 more source

Atherosclerosis in frailty: Not frailty in atherosclerosis [PDF]

open access: yesAtherosclerosis, 2017
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GUARALDI, Giovanni, Raggi, Paolo
openaire   +2 more sources

Cholesteryl ester transfer protein and high density lipoprotein responses to cholesterol feeding in men: relationship to apolipoprotein E genotype.

open access: yesJournal of Lipid Research, 1993
The apolipoprotein (apo) E isoform is an important determinant of the plasma lipoprotein distribution of apoE and of the metabolism of apoE-containing lipoproteins.
LJ Martin   +9 more
doaj   +1 more source

Atherosclerosis and Autoimmunity [PDF]

open access: yesMediators of Inflammation, 2018
Abstract Despite a decline in mortality rates in several countries, cardiovascular disease remains the leading cause of death responsible for approximately half of all deaths on the continent. Coronary artery disease and stroke are often the result of acute complications of atherosclerosis (AS) and atherothrombosis.
Francesca Romana Spinelli   +4 more
openaire   +3 more sources

Inflammation and Atherosclerosis [PDF]

open access: yesEuropean Journal of Vascular and Endovascular Surgery, 2006
The aim of this article is to discuss the role of inflammation in atherosclerosis.An initial chemical, mechanical or immunological insult induces endothelial dysfunction. This triggers a cascade of inflammatory reactions, in which monocytes, macrophages, T lymphocytes and vascular smooth muscle cells participate. Leukocyte adhesion molecules, cytokines,
Kaperonis, EA   +4 more
openaire   +3 more sources

Apolipoproteins C-III and E in apoB- and non-apoB-containing lipoproteins in two populations at contrasting risk for myocardial infarction: the ECTIM study. Etude Cas Témoins sur 'Infarctus du Myocarde

open access: yesJournal of Lipid Research, 1996
Apolipoprotein (apo) C-III and apoE are components of two major classes of plasma lipoproteins, i.e., apoB- and non-apoB-containing lipoproteins. To analyze the relationship between the distribution of apoC-III and apoE among lipoproteins and coronary ...
G Luc   +7 more
doaj   +1 more source

2016 ESC/EAS GUIDELINES FOR THE MANAGEMENT OF DYSLIPIDAEMIAS

open access: yesРоссийский кардиологический журнал, 2017
The Task Force for the Management of Dyslipidaemias of the European Society of Cardiology (ESC) and European Atherosclerosis Society (EAS)Developed with the special contribution of the European Assocciation for Cardiovascular Prevention & ...
Alberico L. Catapano   +18 more
doaj   +1 more source

Body temperature and mouse scoring systems as surrogate markers of death in cecal ligation and puncture sepsis

open access: yesIntensive Care Medicine Experimental, 2018
Background Despite increasing ethical standards for conducting animal research, death is still often used as an endpoint in mouse sepsis studies. Recently, the Murine Sepsis Score (MSS), Mouse Clinical Assessment Score for Sepsis (M-CASS), and Mouse ...
Safiah H. C. Mai   +7 more
doaj   +1 more source

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