Results 191 to 200 of about 68,566 (325)
Cancer Innovation, Volume 4, Issue 4, August 2025.Glioblastoma (GBM) remains a highly malignant brain tumor with limited treatment efficacy, driving the need for innovative cellular immune theranostic strategies that integrate diagnostic and therapeutic approaches. Recent advances in cellular immunotherapy for GBM include chimeric antigen receptor (CAR)‐T/NK cells, macrophage‐based therapies ...
Ying Gong +14 more
wiley +1 more source
Erythema annulare centrifugum and Hodgkin lymphoma during pregnancy
JDDG: Journal der Deutschen Dermatologischen Gesellschaft, EarlyView.
Valentine Horiot +7 more
wiley +1 more source
Comprehensive Physiology, Volume 15, Issue 4, August 2025.Endothelial glycocalyx may be the missing link between angiogenic factors, inflammatory regulation, and endothelial maternal dysfunction in early‐onset preeclampsia and HELLP syndrome. ABSTRACT The pathophysiology of preeclampsia and HELLP syndrome relies on systemic vascular endothelial dysfunction, resulting from angiogenic imbalance due to abnormal ...
Anthony Atallah +4 more
wiley +1 more source
Structural and functional studies in C1q deficiency. [PDF]
, 1982 Rose Marie Chapuis +3 more
openalex +1 more source
FABP7 Expression Modulates the Response of Astrocytes to Induced Endotoxemia
Glia, Volume 73, Issue 8, Page 1627-1641, August 2025.Main Points FABP7 silencing reduces inflammatory response in astrocytes. FABP7 silencing decreases astrocyte‐mediated neurotoxicity. FABP7 silencing attenuates NF‐κB‐dependent transcriptional response during endotoxemia. ABSTRACT Fatty acid binding proteins (FABPs) are a family of small proteins involved in fatty acid (FA) subcellular trafficking.
Mariana Bresque +6 more
wiley +1 more source
Immunochemical and functional analysis of a complete C1q deficiency in man: evidence that C1r and C1s are in the native form, and that they reassociate with purified C1q to form macromolecular C1. [PDF]
, 1980 Michael Loos +4 more
openalex +1 more source
Glia, Volume 73, Issue 8, Page 1746-1766, August 2025.Main Points Microglial NF‐kB signaling increases M1‐like spinal microglia over time after spinal cord injury. Microglial NF‐kB signaling broadly affects gene expression in spinal cord. Microglial NF‐kB signaling modulates splenocytes acutely after spinal cord injury.
Micaela L. O'Reilly +8 more
wiley +1 more source
Inhibition of the activation of Hageman factor (factor XII) by complement subcomponent C1q. [PDF]
, 1987 Esther H. Rehmus +3 more
openalex +1 more source
PI3K/AKT/mTOR Axis in Cancer: From Pathogenesis to Treatment
MedComm, Volume 6, Issue 8, August 2025.The PI3K/AKT/mTOR signaling axis is crucial in cancer progression, regulating cell growth, survival, and metabolism. Often hyperactivated through mutations or loss of tumor suppressors such as PTEN, this pathway promotes cancer cell proliferation and survival by inhibiting apoptosis and activating mTOR, which drives protein synthesis and suppresses ...
Mingyang Jiang +10 more
wiley +1 more source
The metabolism of C1 inhibitor and C1q in patients with acquired C1-inhibitor deficiency
, 1986 J METARNED +3 more
openalex +1 more source