Results 151 to 160 of about 37,904 (214)

Complement Dysregulation in Kidney Diseases: Mechanisms, Biomarkers, and Emerging Targeted Therapies. [PDF]

Int J Mol Sci
Jesiołowski P, Krzywda M, Furmańczyk-Zawiska A, Durlik M.   +3 more
europepmc   +1 more source

Optimizing cohort criteria for multi-country analysis of women experiencing menopause in administrative databases. [PDF]

BMC Med Res Methodol
Banks V, Brash J, Caetano C, Dinkel-Keuthage C, Herrera R, Janssenswillen C, Saadedine M, Schoof N, Moeller C.   +8 more
europepmc   +1 more source

Therapeutic IgG- and IgM-specific proteases disarm the acetylcholine receptor autoantibodies that drive myasthenia gravis pathology. [PDF]

Proc Natl Acad Sci U S A
Bayer AC, Sanmarco LM, Pellerin A, Masi G, Plasencia A, Anderson JM, Nowak RJ, Damato V, Massacesi L, Pham MC, Khani-Habibabadi F, Vital H, Higginson-Scott N, Otipoby KL, Xing Y, Mascanfroni ID, O'Connor KC.   +16 more
europepmc   +1 more source

Complement-Mediated Kidney Diseases: Role of Alternative Pathway in Glomerular Inflammation. [PDF]

Kidney Int Rep
Barratt J, Garred P, Lafayette RA, Zhang H, Floege J.   +4 more
europepmc   +1 more source

Complement and inflammasome crosstalk in chronic inflammation. [PDF]

Front Immunol
Janžič L, Kouter K.
europepmc   +1 more source

LACTIN-V (Lactobacillus crispatus CTV-05) leads to microbial and immune modulation in pregnant women at risk of spontaneous preterm birth:FLIP-1 study

Sykes L, Love R, Bayar E, Capuccini K, Correia GdS, Manoli E, Lee Y, Molina BG, Roberts L, Mountain K, Bokun V, Ng S, Terzidou V, Marchesi J, Takats Z, Lagenaur L, Parks T, Lee P, Bennett P, MacIntyre D.   +19 more
europepmc   +1 more source

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Preparation of monoclonal antibodies to C3b by immunization with C3b(i)-Sepharose

Journal of Immunological Methods, 1989
We have prepared and characterized four monoclonal antibodies (MAbs) to human C3b of high specificity and affinity. Our procedure did not require a purified source of C3b for immunization. Instead, C3b and C3bi were deposited on Sepharose 4B via the alternative pathway of complement activation in normal human serum, and this C3b(i)-Sepharose served as ...
William M Sutherland, J C Edberg, Ronald P Taylor   +2 more
exaly   +3 more sources

Activated C3 (C3b) in the nephritic glomerulus

Pediatric Nephrology, 1993
An autoradiographic technique was developed to assess in the nephritic glomerulus the relative amount of C3 which is in the activated form, C3b, compared with the inactivated form, iC3b. Frozen renal biopsy sections from children and young adults with glomerulonephritis were assessed for the C3b fraction of total C3, using a radiolabeled monoclonal ...
Cynthia Pan, C Frédéric Strife, A James McAdams   +2 more
exaly   +3 more sources

Mechanism of action of the C3b inactivator: Requiremen for a high molecular weight cofactor (C3b-C4bINA cofactor) and production of a new C3b derivative (C3b′)

Immunochemistry, 1977
Abstract We investigated the mechanism of cleavage of human C3b by the C3bINA and documented the polypeptide chain structure of the breakdown products. As reportd for C4b cleavage by C4bINA (Shiraishi & Stroud, 1975; Nagasawa et al., 1976), the C3bINA requires a macromolecular protein for limited proteolysis of C3b.
S, Nagasawa, R M, Stroud
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Degradation of C3b by the C3b Inactivator

The Journal of Immunology, 1971
Abstract The C3b Inactivator (C3bINA) is a normal serum β globulin which destroys the activity of cell-bound C3b in immune hemolysis, immune adherence and the enhancement of phagocytosis. C3bINA treatment of EAC14oxy23 prepared with 125I-labeled C3 releases protein-bound radioactivity into the supernatant fluid.
S Ruddy, K F Austen
openaire   +1 more source