The Network of Exosomes miRNA and p-MLC2 Regulatory Pathway Induced Pathological Cardiac Hypertrophy in Vasn Deficient Mice. [PDF]
J Cell Mol MedHuang B +10 more
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A therapeutic leap: how myosin inhibitors moved from cardiac interventions to skeletal muscle myopathy solutions. [PDF]
J Clin InvestBogomolovas J, Chen J.
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A mutation that prevents myosin from overcoming its inhibitions. [PDF]
J Gen PhysiolShort B.
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High-resolution structures of Myosin-IC reveal a unique actin-binding orientation, ADP release pathway, and power stroke trajectory. [PDF]
Proc Natl Acad Sci U S AChavali SS +4 more
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Cardiac Myosin Inhibitors in Hypertrophic Cardiomyopathy: From Sarcomere to Clinic. [PDF]
Int J Mol SciNakamura K +7 more
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The filopodial myosin DdMyo7 is a slow, calcium-regulated motor. [PDF]
J Biol ChemEddington C, Titus MA.
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Homologous mutations in human β, embryonic, and perinatal muscle myosins have divergent effects on molecular power generation. [PDF]
Proc Natl Acad Sci U S ALiu C +8 more
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The order of things: phosphate release or the power stroke, which does actomyosin do first? [PDF]
Front PhysiolDebold EP, Marang CP, Scott BD.
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Length perturbation analysis was used to demonstrate that β-cardiac myosin characteristic rates of detachment and recruitment in the intact myofilament lattice are accelerated by Pi, phosphorylation of cMyBP-C, and the absence of cMyBP-C. The results suggest that cMyBP-C normally slows myosin detachment, including Pi-dependent detachment, and that ...
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