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Cerebral edema and its treatment [PDF]

open access: yesMedicina (Lithuania), 2007
Cerebral edema is a life-threatening condition that develops as a result of an inflammatory reaction. Most frequently, this is the consequence of cerebral trauma, massive cerebral infarction, hemorrhages, abscess, tumor, allergy, sepsis, hypoxia, and ...
Dalia Adukauskiene
exaly   +2 more sources

Cerebral Edema

The Journal of Nervous and Mental Disease, 1977
Great strides have been made in understanding the pathogenesis of cerebral edema. Treatment is usually successful, particularly with the newer modes of management (mannitol, steroids, hyperventilation); however, cerebral edema is occasionally resistant to all modes of therapy.
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Agents for Cerebral Edema

Neurosurgery, 1981
Hyperventilation, ventricular drainage, and mannitol remain the mainstays of the treatment of cerebral edema not amenable to or following surgical therapy. There appears to be good therapeutic rationale for the use of "low-dose" mannitol in more prolonged treatment of intracranial hypertension (Table 5.1). The beneficial effects of steroids, either in "
R A, de los Reyes   +2 more
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Anesthetics and Cerebral Edema

Anesthesiology, 1976
Localized edema follows the freezing of a small area of cerebral cortex. Effects of five subsequent hours of anesthesia on this edema were studied in six groups of six dogs each. Six anesthetic techniques were studied. In six additional "awake" dogs, anesthesia (halothane) was discontinued immediately after the lesion was made.
A L, Smith, J J, Marque
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High altitude cerebral edema

Neurosurgery, 1986
Acute mountain sickness (AMS) is usually a benign and self-limited illness that befalls previously healthy individuals who ascend rapidly to high altitude without sufficient acclimatization. In its more severe forms, AMS can progress to a life-threatening condition in which pulmonary or cerebral edema can occur singly or in concert.
A J, Hamilton, A, Cymmerman, P M, Black
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Cerebral edema

Seminars in Nephrology, 2001
Two major types of brain edema may be discriminated, characterized by intra- or extracellular fluid accumulation. Intracellular (cytotoxic) edema is found after cerebral ischemia, trauma, intoxications, and metabolic disorders. Pathogenetic mechanisms include (1) failure of active Na+ export via Na/K-ATPase because of energy shortage, (2) increased Na+-
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