Results 111 to 120 of about 256,540 (247)

Combined Inflammatory and Metabolic Defects Reflected by Reduced Serum Protein Levels in Patients with Buruli Ulcer Disease [PDF]

, 2014
Buruli ulcer is a skin disease caused by Mycobacterium ulcerans that is spreading in tropical countries, with major public health and economic implications in West Africa.
Reid Oldenburg (550232), Landier Jordi, Niang, Fatoumata, Sarfo, FS, Sarfo, Fred S, Richard O. Phillips (347896), Mark Wansbrough-Jones, Phillips, Richard O., Forson Mark, Fatoumata Niang (316230), Caroline Demangel (239243), Forson, M, Arnaud Fontanet (157942), Fatoumata Niang, Mark Forson, Niang, F, Oldenburg Reid, Richard O Phillips, Fred S. Sarfo (347891), Jordi Landier (197677), Sarfo Fred S., Fred S Sarfo, Demangel, Caroline, Landier, Jordi, William Thompson, Niang Fatoumata, Demangel, C, Wansbrough-Jones, Mark, Kabiru Abass (550234), Arnaud Fontanet, Wansbrough-Jones Mark, Frimpong, M, Oldenburg, Reid, Richard O. Phillips, William Thompson (241605), Phillips, Richard O, Phillips Richard O., Thompson William, Mark Forson (550235), Sarfo, Fred S., Forson, Mark, Michael Frimpong (550233), Landier, J, Oldenburg, R, Caroline Demangel, Fontanet, A, Frimpong Michael, Abass Kabiru, Fontanet Arnaud, Abass, K, Michael Frimpong, Abass, Kabiru, Frimpong, Michael, Phillips, RO, Wansbrough-Jones, M, Fontanet, Arnaud, Demangel Caroline, Reid Oldenburg, Thompson, William, Mark Wansbrough-Jones (347902), Thompson, W, Kabiru Abass, Jordi Landier, Fred S. Sarfo   +63 more
core   +2 more sources

Cyclic azapeptide CD36 ligand attenuates cardiac injury and reduces long‐chain fatty acid accumulation after myocardial ischemia–reperfusion in mice

FEBS Open Bio, EarlyView.
In a murine model of myocardial ischemia and reperfusion (MI/R), the CD36 azapeptide ligand MPE‐298 reduces cardiac injury and transiently lowers left ventricular long‐chain fatty acids (LCFAs) accumulation 3 h after reperfusion, accompanied by a decrease of oxidative stress and inflammation‐associated genes' expression in the heart and adipose tissue.
Jade Gauvin, Naghme Radmannia, David N. Huynh, Liliane Ménard, Caroline Daneault, Maïté Veilleux, Ahsanullah Ahsanullah, André C. Carpentier, William D. Lubell, Matthieu Ruiz, Huy Ong, Sylvie Marleau, Simon‐Pierre Gravel   +12 more
wiley   +1 more source

Inflammatory mediators, infection, sepsis, and multiorgan failure after severe trauma [PDF]

, 1992
The relation of (multiple) organ failure (OF) to the release of inflammatory mediators and the incidence of infection and sepsis was studied prospectively in 100 patients with multiple trauma (injury severity score=37).
Waydhas, Christian, Trupka, Arnold, Nast-Kolb, Dieter, Schweiberer, Leonhard, Jochum, Marianne, Fritz, Hans, Lenk, Susann, Duswald, Karl-Heimo   +7 more
core  

Genetically Predicted Association of 91 Circulating Inflammatory Proteins with Multiple Sclerosis: A Mendelian Randomization Study [PDF]

Previous studies have validated a close association between inflammatory factors and multiple sclerosis (MS), but their causal relationship is not fully profiled yet.
Peng Wang, Zhiguo Ding, Jingwei Zhou, Shuo Qi, Xin’ai Li, Junhui Wang   +5 more
core   +1 more source

Circulating inflammatory proteins are elevated in type 1 and type 2 diabetes and associated to complications. [PDF]

Diabetes Obes Metab
Abstract Background The presence of low‐grade inflammation has been reported in people with type 2 diabetes and related to the development of (macro)vascular complications. Whether systemic inflammation is present in type 1 diabetes and linked to long‐term complications remains unknown. We used
van Heck JIP, Ajie M, Joosten LAB, Tack CJ, Stienstra R.   +4 more
europepmc   +5 more sources

UiO‐66 metal–organic frameworks in biomedicine: From structural tunability to bioimaging, photodiagnostics, and photodynamic cancer therapy

FEBS Open Bio, EarlyView.
UiO‐66(Zr) metal–organic frameworks are chemically stable, biocompatible, and highly tunable nanomaterials. Their modular structure enables controlled drug delivery, multimodal bioimaging, and light‐activated photodynamic therapy, supporting integrated diagnostic and therapeutic (theranostic) applications in cancer and biomedical research.
Veronika Huntošová, Grigorii Rakhalskii, Miroslav Almáši   +2 more
wiley   +1 more source

Eprodisate for the treatment of renal disease in AA amyloidosis [PDF]

, 2007
Background: Amyloid A (AA) amyloidosis is a complication of chronic inflammatory conditions that develops when proteolytic fragments of serum amyloid A protein (SAA) are deposited in tissues as amyloid fibrils.
Dember, L.M., Hazenberg, B.P.C., Garceau, D., Puéchal, X., Livneh, A., Lesnyak, O., Skinner, Martha, Lesnyak, Olga, Livneh, Avi, Butrimiene, I., Merlini, G., Hauck, Wendy, Obici, L., Dember, Laura M., Gorevic, Peter D., Skinner, M., Butrimiene, Irena, Hawkins, Philip N., Balshaw, Robert, Hazenberg, Bouke P. C.; id_orcid, Lachmann, Helen J., Obici, Laura, Gorevic, P.D., Puechal, Xavier, Hazenberg, Bouke P. C., Hawkins, P.N., Garceau, Denis, Balshaw, R., Lachmann, H.J., Merlini, Giampaolo, Hauck, W.   +30 more
core  

From energy provision to protein synthesis: Tunnelling nanotubes as mediators of intercellular metabolic cooperation in cancer

FEBS Open Bio, EarlyView.
The cytoskeleton‐mediated transport of mitochondria via tunnelling nanotubes restores respiration, increases ATP production, rescues cells from apoptosis, activates the AKT/mTOR signalling pathway, promotes cell migration and invasiveness, contributes to cancer progression and treatment resistance.
Stanislava Martínková, Jan Trnka
wiley   +1 more source

Effects of fatty acids on inflammatory markers studied in vivo and in vitro [PDF]

, 2008
Inflammation involves interactions amongst many different cell types as a defense mechanism of the body. Inflammation is also involved in cardiovascular disease (CVD).
Mohd Yusof, Hayati
core  

The translational relevance of the Annexin A1 pathway in inflammatory pathologies: opportunities for novel therapeutic development [PDF]

, 2009
PhDEndogenous anti-inflammatory mediators form a complex network triggered in the host to dampen cell activation and promote resolution of inflammation.
Dalli, Jesmond
core