Mapping of the continuous epitopes displayed on the Clostridium perfringens type D epsilon-toxin. [PDF]
Braz J Microbiol, 2017 The epsilon toxin, produced by Clostridium perfringens, is responsible for enterotoxemia in ruminants and is a potential bioterrorism agent. In the present study, 15 regions of the toxin were recognized by antibodies present in the serum, with different immunodominance scales, and may be antigen determinants that can be used to formulate subunit ...
Alves GG +4 more
europepmc +5 more sources
Molecular Typing of Clostridium perfringens Isolates from Soil, Healthy, and Diseased Sheep in Egypt by Multiplex PCR [PDF]
Journal of Veterinary Medical Research, 2013 In this study multiplex PCR was used for typing of Clostridium perfringens isolates from soil, clinically healthy and diseased sheep. Clostridium perfringens was isolated from 41 out of 100 soil samples, 12 out of 100 clinically healthy sheep and 118 out
M. Nayel +5 more
doaj +1 more source
Diagnosis of Clostridium perfringens strains isolated from enterotoxemia cases in sheep using polymerase chain reaction (PCR) technique [PDF]
Iraqi Journal of Veterinary Sciences, 2009 This study included diagnosis and typing of 100 bacterial isolates of Clostridium perfringens that isolated from enterotoxaemia cases in sheep in Hama province by using Multiplex PCR.
M. A. Hamad, N. Habra, A. Kalb-Allouz
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Poultry Science, 2021 : Clostridium perfringens (C. perfringens) is the etiological agent of necrotic enteritis and gangrenous dermatitis; 2 diseases that cause significant economic and welfare concerns to the broiler industry.
Courtney A. Fancher +7 more
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Epsilon-Toxin Plasmids of Clostridium perfringens Type D Are Conjugative [PDF]
Journal of Bacteriology, 2007 ABSTRACT Isolates of Clostridium perfringens type D produce the potent epsilon-toxin (a CDC/U.S. Department of Agriculture overlap class B select agent) and are responsible for several economically significant enterotoxemias of domestic livestock.
Meredith L, Hughes +7 more
openaire +2 more sources
Virulence Plasmid Diversity inClostridium perfringensType D Isolates [PDF]
Infection and Immunity, 2007 ABSTRACTClostridium perfringenstype D isolates are important in biodefense and also cause natural enterotoxemias in sheep, goats, and occasionally cattle. In these isolates, the gene (etx) encoding ε-toxin is thought to reside on poorly characterized large plasmids.
Sameera, Sayeed +2 more
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The Pathology of Peracute Experimental Clostridium Perfringens Type D Enterotoxemia in Sheep [PDF]
Journal of Veterinary Diagnostic Investigation, 2004 The pathological findings in sheep with peracute experimental Clostridium perfringens type D enterotoxemia are described. Of 16 animals inoculated intraduodenally with a whole culture of this microorganism and a starch solution in the abomasum, 12 developed clinical signs including increased respiratory efforts, recumbency, paddling, bleating ...
Uzal F.A. +4 more
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mSystems, 2023 Clostridium perfringens is a bacterial species of importance to both public and animal health. The gene optrA is the first gene that confers resistance to the tedizolid, a last-resort antimicrobial agent in human medicine.
Ke Wu +15 more
doaj +1 more source
Genotyping of Clostridium perfringens associated with sudden death in cattle
Journal of Venomous Animals and Toxins including Tropical Diseases, 2009 Toxigenic types of Clostridium perfringens are significant causative agents of enteric disease in domestic animals, although type E is presumably rare, appearing as an uncommon cause of enterotoxemia of lambs, calves and rabbits.
S Miyashiro, L Baldassi, AFC Nassar
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Incidence of some anaerobic bacteria isolated from chicken meat products with special reference to Clostridium perfringens [PDF]
Benha Veterinary Medical Journal, 2017 Anaerobic spore formers, especially Clostridium perfringens, represent one of the most prevalent bacterial food poisoning outbreaks which mostly related to consumption of contaminated meat and meat products, therefore, a total of 125 random raw and half ...
Nabil E., Zakaria M., Shaltout A.
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