Amino acid sequence of the N-terminal forty-two amino acid residues of the C chain of subcomponent C1q of the first component of human complement [PDF]
, 1977 K.B.M. Reid
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Journal of Fish Diseases, Volume 48, Issue 11, November 2025.ABSTRACT Cardiac viral diseases are among the major causes of economic losses in Atlantic salmon (Salmo salar L.) aquaculture. These include cardiomyopathy syndrome (CMS) caused by piscine myocarditis virus (PMCV) and pancreas disease (PD) caused by Atlantic salmonid alphavirus (SAV).
Francesca Riva +14 more
wiley +1 more source
The Role of C1q in the Internal Activation of the First Component of Complement (C1): Studies in Serum of a Selective C1q-Deficient Patient [PDF]
, 1978 Michael Loos +2 more
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Matrix: a complex amalgam of structures and functions in tumor microenvironment
FEBS Open Bio, Volume 15, Issue 10, Page 1552-1569, October 2025.The matrix is a dynamic, intricate three‐dimensional mesh of biomolecules with both structural and functional properties. This review deals with the complexity of this ‘molecular amalgam’ in the tumor microenvironment and highlights its importance in the maintenance and evolution of tumors by describing certain matrix biomolecules, such as ...
Spyros S. Skandalis +3 more
wiley +1 more source
ACR Open Rheumatology, Volume 7, Issue 10, October 2025.Objective Antiphospholipid antibody (aPL) syndrome (APS) classification requires a thrombotic event and detection of lupus anticoagulant (LAC), anticardiolipin antibodies (aCL), or anti–β2‐glycoprotein I (anti‐β2GPI) antibodies on two occasions ≥12 weeks apart. Here, we investigated the utility of anti–phosphatidylserine/prothrombin complex (anti‐PS/PT)
Christina Donath +4 more
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Activation of the first component of complement, C1, by a monoclonal antibody recognizing the C chain of C1q. [PDF]
, 1984 Hans‐Peter Heinz +3 more
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Type I Interferon Signaling Augments Autoimmunity in Neuromyelitis Optica Spectrum Disorder
Advanced Science, Volume 12, Issue 38, October 13, 2025.The cGAS‐STING innate immune pathway is excessively activated both in peripheral monocytes and CNS microglia in patients with NMOSD, leading to an overproduction of IFN‐I. IFN‐I subsequently acts on AQP4‐specific autoreactive T cells. Blocking this pathway may offer novel therapeutic options for individuals with NMOSD.
Tian‐Xiang Zhang +13 more
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Human monoclonal IgG isotypes differ in complement activating function at the level of C4 as well as C1q. [PDF]
, 1988 C Bindon +3 more
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Modulation of Aging Diseases via RAGE Targets: A Dietary Intervention Review
Advanced Science, Volume 12, Issue 38, October 13, 2025.This paper analyzes RAGE ligand‐binding mechanisms and signaling, proposing intervention strategies targeting these interactions. It emphasizes dietary polyphenols, polysaccharides, and terpenoids delivered via functional foods for anti‐aging. These compounds offer long‐term, safe intervention through dietary supplementation, providing dual nutritional
Qian Wu +7 more
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Biosynthesis in vitro of complement subcomponents C1q, C1s and C1̅ inhibitor by resting and stimulated human monocytes [PDF]
, 1983 Jean‐Claude Bensa +2 more
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