Results 181 to 190 of about 18,739,280 (231)

Alteration of reactive oxygen species master transcription factor Nrf2 in keratinocytes exposed to monoclonal pathogenic antibody AK23 against desmoglein-3 in pemphigus vulgaris

Faris M Alabeedi
openalex   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Atypical pemphigus with immunoglobulin G autoantibodies against desmoglein 3 and desmocollin 3

The Journal of Dermatology, 2016
AbstractIn patients with pemphigus vulgaris (PV), pathogenic immunoglobulin (Ig)G antibodies are most commonly directed against desmoglein 3 (Dsg3). It has recently been reported, however, that IgG anti‐desmocollin 3 (Dsc3) antibodies are detected in some cases of pemphigus with or without IgG anti‐Dsg3 antibodies.
K. Kamiya, Y. Aoyama, Chie Wakashima, T. Kudo, K. Nakajima, S. Sano, N. Ishii, K. Teye, T. Hashimoto, K. Iwatsuki, Y. Tokura   +10 more
semanticscholar   +3 more sources

Interleukin‐37 inhibits desmoglein‐3 endocytosis and keratinocyte dissociation via upregulation of Caveolin‐1 and inhibition of the STAT3 pathway

Journal of the European Academy of Dermatology and Venereology, 2023
Pemphigus vulgaris (PV) is a potentially fatal autoimmune bullous disease primarily caused by acantholysis of keratinocytes attributed to pathogenic desmoglein‐3 (Dsg3) autoantibodies.
Junqin Liang, F. Hu, Lidan Mao, Y. Qiu, Fanhe Jiang, Qian Wang, Kailibinuer Abulikemu, Yongzhen Hong, Xinyu Ge, X. Kang   +9 more
semanticscholar   +1 more source

Desmoglein-3 overexpression in oral squamous cell carcinoma is associated with metastasis formation and early recurrence: An immunohistochemical study.

Journal of cranio-maxillo-facial surgery : official publication of the European Association for Cranio-Maxillo-Facial Surgery, 2021
The purpose of this study was to determine the expression patterns of specific desmosomal cadherins (desmogleins [DSG] 1/2/3) in oral squamous cell carcinoma (OSCC), and to examine possible associations with clinicopathological parameters and recurrence ...
M. Troeltzsch, V. Künzel, Selgai Haidari, M. Troeltzsch, S. Otto, M. Ehrenfeld, F. Probst, T. Knösel   +7 more
semanticscholar   +1 more source

Pathogenic human monoclonal antibody against desmoglein 3

Clinical Immunology, 2006
Pemphigus vulgaris (PV) is a potentially fatal autoimmune mucocutaneous disease associated with production of IgG autoantibodies to desmoglein 3 (Dsg3), a 130-kDa epidermal cadherin protein. The binding of pathogenic antibody to Dsg3 on epidermal keratinocytes leads to loss of intercellular adhesion and results in intraepithelial blister formation ...
Shih-Wei, Yeh, Lisa A, Cavacini, Kailash C, Bhol, Mong-Shang, Lin, Mukesh, Kumar, Mark, Duval, Marshall R, Posner, A Razzaque, Ahmed   +7 more
openaire   +2 more sources

Do normal Egyptians possess anti‐desmoglein 3 antibodies?

International Journal of Dermatology, 2014
AbstractBackgroundPemphigus is a group of autoimmune blistering diseases targeting the cell–cell adhesion molecules, desmogleins (Dsgs). Anti‐Dsg antibodies, the hallmark of the disease, were not detected in normal individuals in many populations. In spite of the rarity of pemphigus vulgaris (PV) disease in many parts of the world, PV is not rare in ...
Marwah A, Saleh, Mohamed M, El-Bahy
openaire   +2 more sources

Novel nanoparticle AuNCs conjugated with Desmoglein-3 antibody for FL/CT dual-mode targeted imaging and precise treatment of lung squamous cell carcinoma.

Journal of Colloid and Interface Science
Due to lack of effective, early and non-invasive diagnostic as well as treatment tools, the surgical treatment opportunities for lung squamous cell carcinoma (SCC) are limited, resulting in high mortality rates.
Yujia Zhang, Zhe Yang, Lei Song, Yang Li, Quan Lin   +4 more
semanticscholar   +1 more source

Pathogenic relevance of antibodies against desmoglein 3 in patients with oral lichen planus

Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG
Oral lichen planus (OLP) is a T cell driven disorder that significantly impairs patients’ quality of life. Previous reports suggest that both cellular and humoral activities against desmoglein (dsg) 1 and 3 may be involved in OLP pathogenesis.
D. Didona, M. Schmidt, K. Meier, A. Mesas-Fernández, R. Maglie, E. Antiga, Marisa Klemp, Amir S Yazdi, K. Ghoreschi, Michael Hertl, C. Möbs, F. Solimani   +11 more
semanticscholar   +1 more source

Desmoglein 3 anchors telogen hair in the follicle

Journal of Cell Science, 1998
ABSTRACT Little is known about the function of desmosomes in the normal structure and function of hair. Therefore, it was surprising that mice without desmoglein 3 (the autoantigen in pemphigus vulgaris) not only developed mucous membrane and skin lesions like pemphigus patients, but also developed hair loss.
P J, Koch, M G, Mahoney, G, Cotsarelis, K, Rothenberger, R M, Lavker, J R, Stanley   +5 more
openaire   +2 more sources

Herpetiform pemphigus without anti‐desmoglein 1/3 autoantibodies

The Journal of Dermatology, 2010
AbstractWe report a patient with herpetiform pemphigus who was negative for autoantibodies to desmoglein (Dsg)1 or 3. He had erythemas with vesicles lining the margins on the trunk and extremities. Histopathology revealed intraepidermal blister with prominent eosinophil infiltration.
Hiroko, Nakashima, Manabu, Fujimoto, Rei, Watanabe, Nobuko, Ishiura, Akemi Ishida, Yamamoto, Takashi, Hashimoto, Kunihiko, Tamaki   +6 more
openaire   +2 more sources