Results 21 to 30 of about 2,851 (167)
The Syk tyrosine kinase is required for skin inflammation in an in vivo mouse model of epidermolysis bullosa acquisita [PDF]
, 2017 The inflammatory form of epidermolysis bullosa acquisita is caused by autoantibodies against type VII collagen (C7), a component of the dermal-epidermal junction.
Mócsai, Attila +3 more
core +4 more sources
Epidermolysis bullosa acquisita
Clinics in Dermatology, 2012 Epidermolysis bullosa acquisita (EBA) is a rare, acquired, chronic subepidermal bullous disease of the skin and mucosa characterized by autoantibodies to type VII collagen (C7) structures, a major component of anchoring fibrils, which attach the epidermis to the dermis.
Rishu, Gupta +2 more
openaire +4 more sources
Clinical periodontal diagnosis
Periodontology 2000, EarlyView., 2023 Abstract Periodontal diseases include pathological conditions elicited by the presence of bacterial biofilms leading to a host response. In the diagnostic process, clinical signs such as bleeding on probing, development of periodontal pockets and gingival recessions, furcation involvement and presence of radiographic bone loss should be assessed prior ...
Giovanni E. Salvi +5 more
wiley +1 more source
Epidermolysis bullosa acquisita
Dermatology Online Journal, 2007 A 29-year-old woman presented with 8 months of multiple vesicles, erosions, and milia on the dorsa of her hands and feet. Histopathologic examination demonstrated a subepidermal blister, with a paucity of eosinophils and a lack of blood vessel wall thickening or caterpillar bodies.
Stein, Jennifer A, Mikkilineni, Radha
+8 more sources
Epidermolysis bullosa acquisita
Journal of the European Academy of Dermatology and Venereology, 2013 AbstractEpidermolysis bullosa acquisita (EBA) is a chronic autoimmune subepidermal bullous disease with clinical features similar to the genetic form of dystrophic epidermolysis bullosa. EBA is characterized by the presence of autoantibodies against type VII collagen which is a major component of the anchoring fibrils at the dermal‐epidermal junction ...
J H, Kim, S-C, Kim
openaire +4 more sources
Epidermolysis bullosa acquisita following skin graft in donor and graft sites: A case report
Burns Open, 2023 Epidermolysis bullosa acquisita is a rare mucocutaneous autoimmune blistering disorder which usually presents in adulthood. The disease is mediated by antibodies against type VII collagen, though the precise inciting factors for autoantibody formation ...
Joyce Xia +6 more
doaj +1 more source
Epidermolysis bullosa acquisita
Indian Journal of Dermatology, Venereology and Leprology, 2006 A 20 year old male started developing bubae following even minor trauma on the skin as well as the mucous membranes during the preceding 5 years. The bunae healed with atrophic scars. Involvement of the oral mucous membrane and the eyes, scpcially the right eye, was quite extensive and unusual.
Jayanta Kr, Das +2 more
openaire +2 more sources
Cytoskeletal regulation of inflammation and its impact on skin blistering disease epidermolysis bullosa acquisita [PDF]
, 2016 Actin remodelling proteins regulate cytoskeletal cell responses and are important in both innate and adaptive immunity. These responses play a major role in providing a fine balance in a cascade of biological events that results in either protective ...
Cowin, A., Kopecki, Z., Ludwig, R.
core +2 more sources
Субэпидермальные буллезные дерматозы [PDF]
, 2019 ПЕМФИГОИД БУЛЛЕЗНЫЙПЕМФИГОИД СЛИЗИСТЫХ ОБОЛОЧЕКПРИОБРЕТЕННЫЙ БУЛЛЕЗНЫЙ ЭПИДЕРМОЛИЗДИАГНОСТИКА ДИФФЕРЕНЦИАЛЬНАЯВо второй части статьи, посвященной дерматозам с субэпидермальным расположением пузырей при гистологическом исследовании, описываются буллезный ...
Катина, М. А. +1 more
core +2 more sources
Dermatologica Sinica, 2011 Antiepiligrin cicatricial pemphigoid (AECP) is a chronic autoimmune subepidermal blistering disease characterized by clinical features of cicatricial pemphigoid and circulating IgG antibasement membrane autoantibodies directed against laminin 332.
Chih-Pin Chen +3 more
doaj +1 more source