Results 81 to 90 of about 2,769 (201)

STREPTOCOCCAL FIBRINOLYSIN

open access: yesJournal of Biological Chemistry, 1946
E C, LOOMIS, R M, SMITH
openaire   +2 more sources

MRP8‐Cre Transgenic Mice Are Resistant to Weight Gain and Diet‐Induced Obesity Independently of Neutrophils

open access: yesEuropean Journal of Immunology, Volume 56, Issue 9, September 2026.
The MRP8Cre mouse model is frequently used to investigate the biology of neutrophils. This study reports that MRP8Cre transgenic have a lower body weight and improved metabolic health, especially after high fat diet feeding, which correlates with decreased Serpine1/PAI‐1 expression in metabolic organs. Image created with BioRender.com. ABSTRACT Obesity
Stephan Forisch   +9 more
wiley   +1 more source

Heparan Sulfate Proteoglycans: Master Regulators of Cellular Signaling, Tissue Development, and Neural Function

open access: yesJournal of Neuroscience Research, Volume 104, Issue 9, September 2026.
Ubiquitous cell‐associated, cytoplasmic, synaptic and extracellular matrix HS‐proteoglycans are multifunctional regulators of tissue form and function. HS‐proteoglycans have diverse functions as growth factor co‐receptors in cell proliferation, differentiation, tissue development, mechanical stabilization, synaptogenesis, synaptic plasticity, and ...
James Melrose
wiley   +1 more source

Action of Fibrinolysin (Plasmin) on Proteins.

open access: yesExperimental Biology and Medicine, 1954
SummaryBy the action of fibrinolysin fibrinogen is converted to products which are not coagulated by thrombin, but which are precipitated by TCA. The enzyme is much less active for fibrin and casein. The former is lysed to give a product only about 20% of which is soluble in TCA.
openaire   +2 more sources

Tissue‐Resident Macrophage in Inflammation and Cancer

open access: yesMedComm, Volume 7, Issue 9, September 2026.
Opposing macrophage functions in inflammation and cancer converge on shared mechanistic nodes, informing cross‐disease therapeutic strategies. ABSTRACT Tissue‐resident macrophages (TRMs) are long‐lived immune cells strategically distributed across organs, where their functional plasticity enables both homeostatic maintenance and pathological ...
Siyuan Huang   +13 more
wiley   +1 more source

Characteristics ofGaffkya homari, the Causative Agent of the Lobster Disease Gaffkemia

open access: yes, 1969
Gaffkya homari did not exhibit lipase, protease, or lecithinase exoenzyme activities. The organism was coagulase and fibrinolysin negative; only lactic acid was produced from glucose.
Diane M. Foley   +2 more
core   +1 more source

Bioinformatics Analysis and Experimental Validation of Key Genes Associated With Hypoxia and Ischemia in Myocardial Infarction

open access: yesMolecular Genetics &Genomic Medicine, Volume 14, Issue 9, September 2026.
This study screened hypoxia‐ischemia hub genes for AMI through transcriptomic bioinformatics and animal tests. Merging two GEO datasets yielded 633 DEGs; intersection with GeneCards hypoxia genes produced 21 HIRGs. PPI network and random forest identified core PLAUR and IL1B, whose diagnostic value was validated by GSE66360 via ROC, calibration, and ...
Longsheng Zhang, Ning Liang
wiley   +1 more source

Effect of Growth of Walker 256 Carcinosarcoma on the Fibrinolytic System of the Rat Plasma and Urine

open access: yes, 1970
Growth of a transplanted Walker 256 carcinosarcoma in rats was accompanied by: 1) An increase in plasma fibrinogen and urinary trypsin inhibitors; 2) A significant decrease in plasma plasminogen and hematocrit; 3) A decrease in urokinase activity in ...
Gerda Mootse   +2 more
core   +1 more source

Systemic impact of intramuscular snake envenomation and intravenous immunoglobulin‐based antivenom treatment on murine plasma proteome and peptidome

open access: yesProtein Science, Volume 35, Issue 9, September 2026.
Abstract Snake envenomation remains a critical global public health challenge. In Brazil, the genus Bothrops is responsible for most accidents, typically resulting in coagulopathy, hemorrhage, and renal failure. Despite its clinical prevalence, the molecular mechanisms driving plasma protein disturbances remain poorly understood.
Bianca C. S. C. de Barros   +6 more
wiley   +1 more source

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