Mitochondrial ROS promotes susceptibility to infection via gasdermin D-mediated necroptosis
C. Weindel +16 more
semanticscholar +1 more source
Acute Myocardial Infarction: Molecular Pathogenesis, Diagnosis, and Clinical Management
Key pathogenic mechanisms in AMI. Oxidative stress and mitochondrial dysfunction induce ROS accumulation, while DAMPs‐PRRs interaction triggers inflammatory cascades. Programmed cell death pathways and epigenetic regulation collectively constitute the core components of AMI pathogenesis.
Mengmeng Zhu +5 more
wiley +1 more source
IL-36γ is secreted through an unconventional pathway using the Gasdermin D and P2X7R membrane pores [PDF]
Laura D. Manzanares +8 more
openalex +1 more source
Control of gasdermin D oligomerization and pyroptosis by the Ragulator-Rag-mTORC1 pathway
Charles L. Evavold +10 more
openalex +1 more source
Gasdermin D pores are dynamically regulated by local phosphoinositide circuitry [PDF]
Ana Beatriz Santa Cruz Garcia +3 more
openalex +1 more source
The Card19 locus of murine chromosome 13 regulates terminal cell lysis downstream of caspase activation and Gasdermin-D cleavage [PDF]
Elisabet Bjånes +18 more
openalex
Gasdermin D Deficiency Limits the Transition of Atherosclerotic Plaques to an Inflammatory Phenotype in ApoE Knock-Out Mice [PDF]
Pauline Puylaert +7 more
openalex +1 more source
The killer protein Gasdermin D [PDF]
Clare E. Bryant, John A. Wright
openaire +1 more source

