Results 121 to 130 of about 136,869 (147)

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation. [PDF]

J Cell Biol, 2016
Fine N, Dimitriou ID, Rullo J, Sandí MJ, Petri B, Haitsma J, Ibrahim H, La Rose J, Glogauer M, Kubes P, Cybulsky M, Rottapel R.   +11 more
europepmc   +1 more source

Autophagy suppresses cell migration by degrading GEF-H1, a RhoA GEF. [PDF]

Oncotarget, 2016
Yoshida T, Tsujioka M, Honda S, Tanaka M, Shimizu S.   +4 more
europepmc   +1 more source

Tension on JAM-A activates RhoA via GEF-H1 and p115 RhoGEF. [PDF]

Mol Biol Cell, 2016
Scott DW, Tolbert CE, Burridge K.
europepmc   +1 more source

Upregulation of the GEF-H1 pathway after transient cerebral ischemia. [PDF]

Exp Neurol, 2015
Luo T, Roman P, Liu C, Sun X, Park Y, Hu B.   +5 more
europepmc   +1 more source

Taxane-Induced Conformational Changes in the Microtubule Lattice Activate GEF-H1-Dependent RhoA Signaling

Meiring JCM, Mahapatra V, Gravett MS, Morren D, Saunders HA, Choi SR, José AP, Karhanova A, Metallidou I, Moore A, Spoelstra MF, Liu R, Giono M, Iyer SS, Díaz JF, Lansky Z, Stecker KE, Steinmetz MO, Howes SC, Kapitein LC, Akhmanova A.   +20 more
europepmc   +1 more source

Changes in the GEF-H1 pathways after traumatic brain injury. [PDF]

J Neurotrauma, 2013
Sabirzhanova I, Liu C, Zhao J, Bramlett H, Dietrich WD, Hu B.   +5 more
europepmc   +1 more source

GEF-H1: orchestrating the interplay between cytoskeleton and vesicle trafficking. [PDF]

Small GTPases, 2013
Pathak R, Dermardirossian C.
europepmc   +1 more source

Mechanosensitive FHL2 tunes endothelial function via microtubule-actomyosin crosstalk. [PDF]

EMBO J
Seetharaman S, Devany J, Kim HR, van Bodegraven EJ, Chmiel T, Tzu-Pin S, Chou WH, Fang Y, Gardel ML.   +8 more
europepmc   +1 more source

Vimentin intermediate filaments control actin stress fiber assembly through GEF-H1 and RhoA.

J Cell Sci, 2017
Jiu Y, Peränen J, Schaible N, Cheng F, Eriksson JE, Krishnan R, Lappalainen P.   +6 more
europepmc   +1 more source

The RhoGEF GEF-H1 Is Required for Oncogenic RAS Signaling via KSR-1 [PDF]

Cancer Cell, 2014
SummaryCellular transformation by oncogenic RAS engages the MAPK pathway under strict regulation by the scaffold protein KSR-1. Here, we report that the guanine nucleotide exchange factor GEF-H1 plays a critical role in a positive feedback loop for the ...
Oliver A Kent, David Meiri, Maria-Jose Sandi   +2 more
exaly   +2 more sources