Results 191 to 200 of about 160,364 (357)
Time‐Efficient and Informatic‐Skill‐Light Gap‐Filling for Telomere‐to‐Telomere Genome Assembly
The paper introduces a novel auxiliary software toolbox GapSuite, consisting of two tools Gap‐Aid and Gap‐Graph, which guides users to fill gaps in chromosome‐level genome assembly using sequence‐extension‐based and assembly‐graph‐based strategies. The two tools enable users with limited informatics expertise to efficiently complete gap‐filling on ...
Dong Xu +8 more
wiley +1 more source
Cytokine‐engineered CAR‐T cells represent a promising immunotherapy against malignancies due to direct tumor killing and potent immunity response. However, significant toxicities, including CRS and ICANS, have restricted clinical applications. How to keep the risk‐benefit balance of the advanced therapy is of great importance for maximizing the benefit
Xinru Zhang +7 more
wiley +1 more source
Cell-autonomous regulation of astrocyte activation by the circadian clock protein BMAL1 [PDF]
Cedeno, Michelle R +11 more
core +1 more source
Allele-specific nuclear positioning of the monoallelically expressed astrocyte marker GFAP [PDF]
Takumi Takizawa +4 more
openalex +1 more source
Biomarkers and clinical rules for the management of mild traumatic brain injury: a narrative review. [PDF]
Salgado S +5 more
europepmc +1 more source
Variants in GFAP Associated with Alexander Disease, with links to PubMed
Messing, Albee
openalex +1 more source
Oral nanoCEL exhibits effective intestinal targeting of antigen‐presenting cells and restores the Th17/Treg balance in lymph nodes and spleen, ultimately protecting the blood‐retinal barrier by inhibiting peripheral immune cell infiltration and suppressing retinal glial cell activation.
Jinrun Chen +13 more
wiley +1 more source
Plasma GFAP as a mediator and moderator of Aβ, tau, and neurodegeneration in Alzheimer's disease. [PDF]
Kim JS +12 more
europepmc +1 more source
Cellular Identity Crisis: RD3 Loss Fuels Plasticity and Immune Silence in Progressive Neuroblastoma
Researchers discovered that therapy‐induced loss of RD3 protein in neuroblastoma triggers a dangerous shift: cancer cells become more stem‐like, invasive, and resistant to treatment while evading immune detection. RD3 loss suppresses antigen presentation and boosts immune checkpoints, creating an immune‐silent environment.
Poorvi Subramanian +7 more
wiley +1 more source

