Results 81 to 90 of about 65,627 (289)
Farmakologija receptora i ćelijskih transportera za glutamat [PDF]
Milovanović Dragan R. +1 more
doaj +1 more source
A 3D disease model is developed using customized hyaluronic‐acid‐based hydrogels supplemented with extracellular matrix (ECM) proteins resembling brain ECM properties. Neurons, astrocytes, and tumor cells are used to mimic the native brain surrounding.
Esra Türker +16 more
wiley +1 more source
Near‐Infrared Organic Photovoltaic Electrodes for Subretinal Neurostimulation
Organic photovoltaic electrodes based on the D18:Y6 blend enable precise and light‐controlled activation of retinal ganglion cells in a degenerating retina. NIR Light‐driven activation of retinal ganglion cells, tunable stimulation parameters, and biocompatibility with human retinal organoids highlight their potential for next‐generation prosthetics ...
Andrea Corna +10 more
wiley +1 more source
Osnovi glutamatergičke neurotransmisije [PDF]
Milovanović Dragan R. +1 more
doaj +1 more source
Human glutaminyl cyclase and bacterial zinc aminopeptidase share a common fold and active site
Background Glutaminyl cyclase (QC) forms the pyroglutamyl residue at the amino terminus of numerous secretory peptides and proteins. We previously proposed the mammalian QC has some features in common with zinc aminopeptidases.
Misquitta Stephanie A +3 more
doaj +1 more source
A unidirectional cerebral organoid–organoid neural circuit is established using a microfluidic platform, enabling controlled directional propagation of electrical signals, neuroinflammatory cues, and neurodegenerative disease–related proteins between spatially separated organoids.
Kyeong Seob Hwang +9 more
wiley +1 more source
Background: Considering the effect of statins on the regulation of dopamine neurotransmitters and glutamates and importance of the treatment of obsessive-compulsive disorder (OCD) due to its relatively high prevalence and disability of available drugs in
Shahla Akouchekian +4 more
doaj +1 more source
Glutamate-Induced Hyperglycemia.
SummaryPlasma glucose rose linearly with increasing amounts of glutamate up to a maximal dose of 0.2 mmoles/100 g body weight. The glutamate effect could not be explained by stoichiometric conversion of glutamate to glucose. Glutamate produced hyper-glycemia after 14-hour fast, at which time glucagon had no effect on glucose concentration ...
R, Marcus, G, Reaven
openaire +2 more sources

