Results 211 to 220 of about 122,606 (315)
Erythrocyte glutathione peroxidase (GSH-Px) polymorphism in
Sandholm M+3 more
doaj +1 more source
Intramitochondrial localization of glutathione-peroxidase (GSH-PO) in the rat adrenocortical cells.
Masanori Murakoshi+3 more
openalex +2 more sources
Copper in cancer: friend or foe? Metabolism, dysregulation, and therapeutic opportunities
Abstract Copper, one of the essential nutrients for the human body, acts as an electron relay in multiple pathways due to its redox properties. Both deficiencies and excesses of copper lead to cellular fragility. Therefore, it can manifest pro‐ and anti‐cancer properties in tumors. Therefore, it is crucial to clarify the copper activity within the cell.
Dan Shan+13 more
wiley +1 more source
Immunocytochemical localization of glutathione-peroxidase (GSH-PO) in rat peritoneal macrophages.
Masanori Murakoshi+2 more
openalex +2 more sources
Combating cancer immunotherapy resistance: a nano‐medicine perspective
Abstract Cancer immunotherapy offers renewed hope for treating this disease. However, cancer cells possess inherent mechanisms that enable them to circumvent each stage of the immune cycle, thereby evading anti‐cancer immunity and leading to resistance.
Xiangyi Kong+10 more
wiley +1 more source
Immunocytochemical localization of glutathione-peroxidase(GSH-PO) in the rat ovary.
Masanori Murakoshi+3 more
openalex +2 more sources
Advances in Cytosolic Delivery of Proteins: Approaches, Challenges, and Emerging Technologies
Proteins regulate almost all biological processes, and overcoming the protein's natural membrane inpermeability will provide a unique opportunity for intracellular targeting of related therapeutic proteins. Chemical modification of proteins and different types of delivery vectors can mediate intracellular transport of proteins, providing new ideas for ...
Wenyan Zhang+8 more
wiley +1 more source
The NADPH oxidase NOX4 regulates redox and metabolic homeostasis preventing HCC progression
Loss of NOX4 in HCC tumor cells induces metabolic reprogramming in a Nrf2/MYC‐dependent manner to promote HCC progression. Abstract Background and Aims The NADPH oxidase NOX4 plays a tumor‐suppressor function in HCC. Silencing NOX4 confers higher proliferative and migratory capacity to HCC cells and increases their in vivo tumorigenic potential in ...
Irene Peñuelas‐Haro+14 more
wiley +1 more source