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Mouse Histocompatibility Antigens

Angewandte Chemie International Edition in English, 1978
AbstractIn this article experimental findings and new theories concerning the chemical structure and biological function of histocompatibility antigens in the immune surveillance of virus‐infected and malignant cells are discussed. Amongst these antigens, the H‐2 antigens of the mouse have been studied most extensively.
exaly   +3 more sources

Histocompatibility Antigens in Leprosy

Vox Sanguinis, 1975
Results of HL-A typing are presented in 82 patients with leprosy and 50 normal Filipinos from Cebu, and 144 normal Filipino immigrants from the Luzon area. Comparisons of HL-A antigen frequencies among the total patients and normals of Cebu showed no statistically significant differences; however, HL-A10 was increased in frequency among the patients ...
G S, Smith   +4 more
openaire   +2 more sources

Minor histocompatibility antigens

Immunology Letters, 1991
Immune responses against foreign tissue or organs can be directed against alloantigenic differences between donor and host encoded by genes of the major histocompatibility complex (MHC; HLA in man and H-2 in mouse). However, when MHC antigens are matched, as in HLA-identical siblings, or between different mouse strains sharing the same H-2 haplotype ...
openaire   +4 more sources

Histocompatability Antigens and Schizophrenia

British Journal of Psychiatry, 1978
The HLA (Human Leucocyte Antigen) types A and B were studied in 80 patients diagnosed as schizophrenic. There was an increased incidence of HLA-BW5 and a decrease in HLA-AW29 and HLA-BW17 as compared with healthy controls. In the sub-group of patients exhibiting Schneider's first-rank symptoms there was an increased incidence of HLA-A1 with a decrease ...
P, McGuffin, A E, Farmer, S M, Rajah
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Medial Histocompatibility Antigens

Scandinavian Journal of Immunology, 1981
Medial histocompatibility (H) antigens are weak H antigens, recognized by unrestricted T cells; they differ thus from both major and minor H antigens. An example, Qed‐1, is described in detail, and other known medial H antigens of the mouse are reviewed.
Fischer, L K, Langhorne, J
openaire   +2 more sources

Minors come of age: minor histocompatibility antigens and graft-versus-host disease [PDF]

open access: yesBiology of Blood and Marrow Transplantation, 2004
Minor histocompatibility antigens (miHA) are responsible for the occurrence of graft-versus-host disease in the setting of a major histocompatibility complex matched sibling allogeneic stem cell transplantation.
Nelson Chao
exaly   +2 more sources

Histocompatibility Antigens and Diabetic Retinopathy

Diabetes, 1977
Of 160 patients with onset of diabetes at or after 30 years of age, the 84 with no evidence of diabetic retinopathy were found to have significantly increased prevalences of HLA-Al and B8 when compared with the 76 with retinal complications or with the 282 healthy blood donors.
B, Becker   +4 more
openaire   +2 more sources

Histocompatibility Antigens and Disease

Archives of Dermatology, 1977
In recent years there has been an extensive search for associations between specific diseases and antigens of the major HLA histocompatibility complex. This search was stimulated by the demonstration of a genetic linkage between the mouse's major histocompatibility complex and resistance to viral-induced leukemias, specific autoimmune diseases, and ...
openaire   +2 more sources

Histocompatibility antigens (HLA) and uveitis

Survey of Ophthalmology, 1980
The studies of histocompatibility (HLA) antigens have contributed to the understanding of the pathogenesis of uveitis. Acute anterior uveitis is associated with rheumatic disorders such as ankylosing spondylitis. HLA-B 27 antigen is present in 98% of these cases. There are genetic and environmental factors influencing the activity of the disease. HLA-B
Y, Scharf, S, Zonis
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Human Histocompatibility Antigens

1983
Numerous chapters in this volume testify to the importance of molecular biology the analysis of problems relating to human cancer. Most recently, it was shown that human (onco) genes carrying single nucleotide substitutions may confer the transformed phenotype rather than encode a presumably normal - albeit unknown - cellular function [1].
openaire   +2 more sources

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