When the biological clock is disordered: circadian rhythm disruption as a core pathophysiological mechanism in anxiety and depression. [PDF]
Yuan J, Zhang J, Yu M, Li L.
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Reproduction under Siege: How Chronic Stress Hijacks the HPG Axis?
Xu D, Zhao X, Chen W, Liu Y, Wang C.
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3-Hydroxypropionic acid converts inflammatory macrophage glycolysis into mitochondrial oxidation through GAPDH carboxyethylation. [PDF]
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Reanalysis data fusion-based tropical cyclone formation prediction network. [PDF]
Hai Y, Peng Y, Wang Y, Cheng Z.
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Mechanistic Study of Schisandrin B in Alleviating Anxiety-Like Behaviors in Rats via Enhancement of Hippocampal Synaptic Plasticity and Restoration of the Intestinal Barrier. [PDF]
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Chronic stress, cortisol dysregulation, and neurodegenerative vulnerability: mechanistic pathways linking HPA-axis dysfunction to Alzheimer's disease risk. [PDF]
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DNA sequencing-based typing of HPA-1 to HPA-17w systems
International Journal of Hematology, 2008Although several DNA-based human platelet antigens (HPA) typing techniques, such as PCR-SSP and PCR-SSO, have been established, the typing errors and the lack of interlaboratory reproducibility are still the issues of concerns. In the present study, polymerase chain reaction primers were designed for identification of all the phenotypically different ...
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This paper outlines the interferences of the most widely used drugs with hypothalamo-pituitary-adrenal function and the related laboratory parameters, with the purpose of providing practical help to clinicians during testing for hypo- or hypercortisolemic states.
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The hormones of the hypothalamus-pituitary-adrenal (HPA) axis influence memory in situations of acute and chronic stress. The present review tries to summarize the current state of knowledge by describing the enhancing as well as the impairing effects of stress or glucocorticoid (GC) treatment documented in animals and humans.
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