Results 51 to 60 of about 20,403 (220)
Animal, 2016 We have previously demonstrated that a sharp rise in feed intake (hyperphagia) and spontaneous liver steatosis could be experimentally induced in domestic Greylag geese by combining a short photoperiod and a sequence of feed restriction followed by ad ...
X. Fernandez +5 more
doaj +1 more source
Gut microbiota as a novel therapeutic target for eating disorders and obesity
British Journal of Pharmacology, EarlyView.The graphical abstract highlights the bidirectional interaction between the gut microbiota and central homeostatic and hedonic mechanisms regulating food intake, which contributes to overweight and obesity. It also illustrates emerging microbiota‐based biomarkers and therapeutic strategies (including prebiotics, probiotics, postbiotics, synbiotics and ...
Solveiga Samulėnaitė +5 more
wiley +1 more source
The FEBS Journal, EarlyView.Circadian disruption via constant light induces sex‐specific remodeling of the microbiome, lipid mediators, and immune responses. Females exhibit broader gut microbiota alterations, reduced jejunal oxylipins, increased hypothalamic N‐acylethanolamines, and a more inflammatory cytokine profile. Muscle responses are sex‐ and fiber type‐dependent: females
Pejman A. Pashaki +7 more
wiley +1 more source
Clinical Case A clinical case of Prader–Willi syndrome
Клинический разбор в общей медицинеRelevance. Prader–Willi syndrome (SPW) is a rare genetic disease associated with a predominant legion of the nervous system with subsequent involvement of other systems.
Aleksandra V. Serezhkina +5 more
doaj +1 more source
SARS-CoV-2 Infection Causes Relapse of Kleine-Levin Syndrome: Case Report and Review of Literature
Neurology International, 2021 Recurrent episodes of hypersomnia, hypersexuality, compulsive eating, behavioral and cognitive disturbances, are the basic clinical features of Kleine-Levin syndrome (KLS).
Marino Marčić +2 more
doaj +1 more source
Altered neural electrophysiological properties in the anterior cingulate cortex in a mouse model of Prader-Willi syndrome. [PDF]
J PhysiolAbstract figure legend Prader‐Willi syndrome (PWS) originates from deficiencies in chromosome 15q11‐13 region clustering around a critical region containing multiple‐repeat non‐coding RNA gene Snord116, resulting in metabolic and behavioural abnormalities leading to hyperphagia and obesity.
Rybalchenko V, Butler R.
europepmc +2 more sources
Ibis, EarlyView.Individual variation in avian migration timing is still poorly understood, yet recent evidence shows that it is closely linked to personality traits and may also be affected by food availability. Here, we examined how the interaction of the personality trait exploration speed and food availability affected the departure timing of Red Knots Calidris ...
Tohar Tal +4 more
wiley +1 more source
Insulin Sensitivity and the Onset of Hyperphagia in Fatty Rats [PDF]
, 2004 Hyperphagia emerges suddenly after 22 days of age in Zucker fatty (fa/fa) rats, and fatties become visibly obese within days. This rapid increase in growth rate is likely to be associated with substantial changes in glucose metabolism. The objective of
Durham, Holiday Agnes
core
Mode of action of DCCR in hyperphagia.
, 2019 Mode of action of DCCR in hyperphagia.
Virginia Kimonis (7418696) +4 more
core +1 more source
Virus-Induced Voracity: Uncovering Hyperphagia Post-Herpes Simplex Virus Type 1
Case Reports in NeurologyIntroduction: Herpes simplex virus type 1 (HSV-1) is the leading cause of sporadic fatal encephalitis, typically presenting with temporal lobe abnormalities.
Arpan Mitra +5 more
doaj +1 more source