Results 21 to 30 of about 149,546 (225)

The Treatment of IgA Nephropathy [PDF]

open access: yesKidney Diseases, 2015
<b><i>Background:</i></b> IgA nephropathy (IgAN) is a very common glomerulonephritis worldwide. Nevertheless, treatment options for primary IgAN are still largely based on opinion or weak evidence. There is a lack of large randomized controlled trials (RCT) that provide a definitive immunosuppressive protocol for IgAN.
Leung, JCK, Lai, KN, Tang, SCW
openaire   +4 more sources

IgA nephropathy in a patient receiving infliximab for generalized pustular psoriasis

open access: yesBMC Nephrology, 2020
Background IgA nephropathy is the most common glomerulonephritis. Secondary IgA nephropathy complicated with systemic diseases, including psoriasis, is also often reported.
Yuka Segawa   +9 more
doaj   +1 more source

Integration of three machine learning algorithms identifies characteristic RNA binding proteins linked with diagnosis, immunity and pyroptosis of IgA nephropathy

open access: yesFrontiers in Genetics, 2022
Objective: RNA-binding proteins (RBPs) are essential for most post-transcriptional regulatory events, which exert critical roles in nearly all aspects of cell biology.
Xueqin Zhang   +7 more
doaj   +1 more source

IgA nephropathy in a patient with ankylosing spondylitis well controlled with etanercept [PDF]

open access: yesKosin Medical Journal, 2018
Ankylosing spondylitis (AS) can involve the eye, gastrointestinal system, cardiopulmonary system, skin, kidneys, and spinal and peripheral joints. It is rarely accompanied by immunoglobulin A (IgA) nephropathy. Although IgA is involved in both AS and IgA
Do-Hyeong Lee   +3 more
doaj   +1 more source

Aspirin alleviates the symptoms of immunoglobulin A nephropathy via suppressing platelets-mediated non-canonical NF-κB activation in B cells

open access: yesSTEMedicine, 2023
Purpose: Antiplatelet aggregation drugs, such as aspirin, can alleviate pathological renal damage in immunoglobulin A (IgA) nephropathy, although the precise mechanism is unclear.
Nomvula Johannes, Priyanka Maunga
doaj   +1 more source

IgA Nephropathy [PDF]

open access: yesJournal of the American Society of Nephrology, 2000
IgA nephropathy (IgAN), a mesangial proliferative glomerulonephritis (GN), is the most common GN in all parts of the world where renal biopsy is widely practiced. It is unique among glomerular diseases in being defined by immunohistochemical findings, i.e., mesangial deposition of IgA, rather than by light microscopy.
Jürgen, Floege, John, Feehally
openaire   +2 more sources

Use of gas chromatography mass spectrometry to elucidate metabolites predicting the phenotypes of IgA nephropathy in hyper IgA mice.

open access: yesPLoS ONE, 2019
IgA nephropathy, a common chronic kidney disease, has various possible outcomes. Therefore, the identification of novel prognostic biomarkers is needed.
Makoto Kurano, Yutaka Yatomi
doaj   +1 more source

IgA Glycosylation in IgA Nephropathy [PDF]

open access: yes, 1998
IgA nephropathy (IgAN) is the commonest form of glomerulonephritis in developed countries where renal biopsy is widely practiced1. IgAN is defined by the deposition of IgA in the glomerular mesangium. Most typically, patients present with episodes of visible haematuria, which often concide with respiratory or gastrointestinal infection.
A, Allen, J, Feehally
openaire   +2 more sources

Secondary IgA nephropathy [PDF]

open access: yesKidney International, 2018
IgA nephropathy is the most common primary glomerulonephritis worldwide. Its frequent coexistence with inflammatory, infectious, or malignant processes raises the possibility of a pathologic rather than coincidental association. Major strides have been made to elucidate the underlying pathophysiologic events that culminate in the development of primary
Manish K, Saha   +3 more
openaire   +2 more sources

Murine models of renal disease: Possibilities and problems in studies using mutant mice [PDF]

open access: yes, 2000
The elucidation of the pathogenesis of human renal disease at the molecular level has been facilitated by the growing field of gene targeting and the development of mouse strains with single-gene deletions - the `knock-out' mice. Experimental nephrology,
Anders, Hans-Joachim   +1 more
core   +1 more source

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