Results 261 to 270 of about 1,145,138 (308)

Serum amyloid A: The “other” inflammatory protein

Current Atherosclerosis Reports, 2006
Inflammation long has been recognized as a hallmark of atherosclerotic lesions, but more recently attention has focused on chronic low-level elevations of specific plasma inflammatory proteins such as C-reactive protein (CRP) and serum amyloid A (SAA), which may not only represent markers of atherosclerosis risk but also participate directly in ...
Kevin D, O'Brien, Alan, Chait
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Assays for macrophage inflammatory proteins

1997
Publisher Summary This chapter describes methodology to provide the basic experimental grounding in two clinically relevant areas of chemokine research: the regulation of hematopoietic progenitor cell proliferation and the mechanism of human immunodeficiency virus (HIV) infection.
A M, Buckle, S, Craig, L G, Czaplewski
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Acute phase proteins in the monitoring of inflammatory disorders

Baillière's Clinical Rheumatology, 1994
The acute phase reaction is in most circumstances a good indicator of (local) inflammatory activity and tissue damage. CRP is a direct and quantitative measure for the acute phase reaction and due to its fast kinetics provides adequate information of the actual situation.
VANLEEUWEN, MA, VANRIJSWIJK, MH
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Regulation of Inflammatory Proteins

2014
Inflammation involves a complex interplay of immunological responses in reaction to an infection from invading pathogens and to tissue injury. However, if the inflammatory stimulus is not eliminated or if there is an aberrant response, chronic inflammation and disease can result.
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Heat Shock Proteins and the Inflammatory Response

Annals of the New York Academy of Sciences, 1998
ABSTRACT: The heat shock proteins (HSPs) are important in the cellular response to stress and in cellular homeostatic functions such as protein synthesis and protein transport across membranes. An emerging body of data supports a role for HSPs in the inflammatory response, suggesting that HSPs participate in cytokine signal transduction and in the ...
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Model Structure for the Inflammatory Protein C5a

Science, 1985
The complement cleavage product C5a is a potent stimulant of inflammatory processes; thus, inhibition of C5a activity is of therapeutic interest. The three-dimensional structure of the major portion of C5a was modeled from the homologous C3a crystal structure by comparative modeling techniques.
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Fuzzy logic and inflammatory protein variations

Clinica Chimica Acta, 1998
This tutorial style paper has been written for physicians and biologists who are not necessarily familiar with fuzzy set theory and biomedical applications. The field is introduced in the framework of medical diagnosis problems and illustrated with an application to inflammatory protein variations.
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The Acute Phase Protein Profile in Inflammatory Arthropathies

Scandinavian Journal of Rheumatology, 1987
A quantitative evaluation of the changes of the levels of acute phase reactants (APRs) during inflammatory phenomena is a useful tool both in monitoring the course of some inflammatory diseases and the efficacy of the anti-inflammatory therapy. Nonetheless, the information collected through the various indices appears unable to provide either an ...
LAPADULA, Giovanni, AND NUMO R.
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Tumor promoters: from chemicals to inflammatory proteins

Journal of Cancer Research and Clinical Oncology, 2013
The classic two-stage chemical carcinogenesis in rodents is not directly linked to multistage carcinogenesis in humans. In light of our findings that tumor necrosis factor-α (TNF-α) is an endogenous tumor promoter and that TNF-α-inducing protein (Tipα) of Helicobacter pylori stimulates progression of cancer and epithelial-mesenchymal transition, we ...
Hirota, Fujiki, Eisaburo, Sueoka, Masami, Suganuma   +2 more
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Anti-inflammatory peptides and proteins in inflammatory bowel disease.

Current opinion in investigational drugs (London, England : 2000), 2002
Current therapy of inflammatory bowel disease, ie, ulcerative colitis and Crohn's disease, is neither sufficient nor disease-modifying. Long-term treatment with non-specific antiinflammatory drugs aminosalicylates, corticosteroids and immunosuppressants is often accompanied with undesirable and potentially serious side effects.
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