Results 11 to 20 of about 49,064 (119)

Connexin mimetic peptides reversibly inhibit Ca2+ signaling through gap junctions in airway cells

open access: yes, 2000
The effect of peptides with sequences derived from connexins, the constituent proteins of gap junctions, on mechanically stimulated intercellular Ca(2+) signaling in tracheal airway epithelial cells was studied.
Boitano, Scott, Evans, William Howard
core   +6 more sources

The translational relevance of the Annexin A1 pathway in inflammatory pathologies: opportunities for novel therapeutic development [PDF]

open access: yes, 2009
PhDEndogenous anti-inflammatory mediators form a complex network triggered in the host to dampen cell activation and promote resolution of inflammation.
Dalli, Jesmond
core   +4 more sources

Elucidation of a molecular mechanism controlling inflammation during bacterial infection [PDF]

open access: yes, 2015
Recently we described a mechanism of gap junction-mediated communication between infected and uninfected epithelial cells that potentiates innate immunity during infection by the enteroinvasive bacterium Shigella flexneri.
Tschon, Therese
core   +1 more source

CLE peptide signaling during plant development [PDF]

open access: yes, 2009
Peptide signaling in plants is a rapid developing area of research which focuses on so called peptide hormones. These signaling molecules are utilized for inter-cellular communication in different developmental processes, beside the usage of the more ...
Fiers, M.A.   +3 more
core   +1 more source

Specific integrin alpha and beta chain phosphorylations regulate LFA-1 activation through affinity-dependent and -independent mechanisms [PDF]

open access: yes, 2005
Integrins are adhesion receptors that are crucial to the functions of multicellular organisms. Integrin-mediated adhesion is a complex process that involves both affinity regulation and cytoskeletal coupling, but the molecular mechanisms behind this ...
Susanna C. Fagerholm   +11 more
core   +1 more source

Storage-induced changes in erythrocyte membrane proteins promote recognition by autoantibodies [PDF]

open access: yes, 2012
Contains fulltext : 109602.pdf (Publisher’s version ) (Open Access)Physiological erythrocyte removal is associated with a selective increase in expression of neoantigens on erythrocytes and their vesicles, and subsequent autologous ...
Dinkla, S.   +19 more
core   +3 more sources

Host protein phosphorylation during "Shigella flexneri" infection : a phosphoproteomic based systems biology approach [PDF]

open access: yes, 2015
The enteroinvasive bacterium Shigella flexneri triggers its uptake into epithelial cells by injecting several effector proteins via its type three secretion system (TTSS) and interferes with various host cell processes at later stages of infection.
Schmutz, Christoph
core   +1 more source

Desmoglein 2 is less important than desmoglein 3 for keratinocyte cohesion. [PDF]

open access: yes, 2013
Desmosomes provide intercellular adhesive strength required for integrity of epithelial and some non-epithelial tissues. Within the epidermis, the cadherin-type adhesion molecules desmoglein (Dsg) 1-4 and desmocollin (Dsc) 1-3 build the adhesive core of ...
Bettina Kempf   +17 more
core   +2 more sources

In silico analyses of conservational, functional and phylogenetic distribution of the LuxI and LuxR homologs in Gram-positive bacteria

open access: yesScientific Reports, 2017
LuxI and LuxR are key factors that drive quorum sensing (QS) in bacteria through secretion and perception of the signaling molecules e.g. N-Acyl homoserine lactones (AHLs).
Akanksha Rajput, Manoj Kumar
doaj   +1 more source

Expressions of Transforming Growth Factor β1 Signaling Cytokines in Aortic Dissection

open access: yesBrazilian Journal of Cardiovascular Surgery
Objective: To demonstrate the underlying mechanisms of aortic dissection compared to those of coronary artery disease in terms of the transforming growth factor-beta (TGF-β) signaling pathway.
Shi-Min Yuan, Hong Lin
doaj   +1 more source

Home - About - Disclaimer - Privacy