Results 61 to 70 of about 2,108,740 (316)

Role of IL-33 and ST2 signalling pathway in multiple sclerosis: expression by oligodendrocytes and inhibition of myelination in central nervous system [PDF]

open access: yes, 2016
Recent research findings have provided convincing evidence indicating a role for Interleukin-33 (IL-33) signalling pathway in a number of central nervous system (CNS) diseases including multiple sclerosis (MS) and Alzheimer’s disease.
Allan, Debbie   +7 more
core   +2 more sources

Characterization of Endothelial Cells Associated with Hematopoietic Niche Formation in Humans Identifies IL-33 As an Anabolic Factor [PDF]

open access: yes, 2018
Bone marrow formation requires an orchestrated interplay between osteogenesis, angiogenesis, and hematopoiesis that is thought to be mediated by endothelial cells.
Adisty, M.N. (Maria)   +13 more
core   +2 more sources

Role of interleukins in heart failure with reduced ejection fraction

open access: yesAnatolian Journal of Cardiology, 2019
Heart failure (HF) is the leading cause of morbidity and mortality in developed countries, and it is the primary cause of mortality in the elderly worldwide.
Oliwia Anna Segiet   +4 more
doaj   +1 more source

IL-25 and IL-33 Serum Levels are not Associated with the Type of Allergen Causing Allergic Rhinitis [PDF]

open access: yesIranian Journal of Immunology, 2019
Background: Fungal aeroallergens might sensitize the airway which in turn produces a specific cytokine profile. Objective: To evaluate the IL-25 and IL-33 profile in patients with fungal allergic rhinitis. Methods: The present study examined patients who
Reza Abbasi   +6 more
doaj   +1 more source

Interleukin 33 expression in human arthritis [PDF]

open access: yesAnnals of the Rheumatic Diseases, 2011
Background and objectives Interleukin 33 (IL-33) is the most recently discovered member of the IL-1 family of cytokines. Recent evidence in human and mouse suggests a role for IL-33 and its receptor T1/ST2 in arthritis. In this study, the authors quantified IL-33 levels in serum and synovial fluid (SF), and assessed synovial IL-33 expression levels ...
Dominique Talabot-Ayer   +6 more
openaire   +1 more source

Alarmins in frozen shoulder: a molecular association between inflammation and pain [PDF]

open access: yes, 2017
Background: The pathophysiological mechanisms behind proliferation of fibroblasts and deposition of dense collagen matrix in idiopathic frozen shoulder remain unclear.
Akbar, Moeed   +10 more
core   +1 more source

A Multi-marker Model for Predicting Decompensated Heart Failure in Patients with Prior Acute Myocardial Infarction [PDF]

open access: yes, 2020
The aim of the study was to assess the prognostic value of determining the plasma concentration of NT-proBNP and ST2 in the patients with decompensated HF and prior acute myocardial infarction and their combination in this category of patients.Materials ...
Levandovska, K. (Khrystyna)
core   +3 more sources

Cellular and clinicopathological features of the IL-33/ST2 axis in human esophageal squamous cell carcinomas

open access: yesCancer Cell International, 2018
Background Emerging evidence has suggested that interleukin (IL)-33 and its primary functional receptor ST2 are involved in the pathogenesis of tumorigenesis.
Guanglin Cui   +5 more
doaj   +1 more source

CDX2 regulates interleukin‐33 gene expression in intestinal epithelial cells (LS174T)

open access: yesFEBS Open Bio, 2021
Dysregulation of interleukin‐33 (IL‐33) has been implicated in the pathogenesis of several autoimmune and inflammatory diseases, but few studies have examined transcriptional regulation of the IL33 gene.
Sylvester Larsen   +8 more
doaj   +1 more source

The early transcriptomic response to interleukin 1β and interleukin 33 in rat neonatal cardiomyocytes [PDF]

open access: yes, 2013
In the heart, inflammatory cytokines including interleukin (IL) 1β are implicated in regulating adaptive and maladaptive changes, whereas IL33 negatively regulates cardiomyocyte hypertrophy and promotes cardioprotection.
Barrett, Oliver P T   +4 more
core   +1 more source

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