Results 231 to 240 of about 4,008,604 (402)
The role of T-cell interleukin-17 in conducting destructive arthritis: lessons from animal models. [PDF]
, 2005 Erik Lubberts +2 more
openalex +1 more source
Advanced Healthcare Materials, EarlyView.A) SLM generates biomimetic bone scaffolds with consistent porosity but varying TPMS‐Gyroid unit cell designs (TG15, TG20, TG25, TG30). B) By enhancing the expression of ITGB1, TPMS‐Gyroid scaffolds can facilitate osteogenic differentiation in BMSCs and promote M2 polarization in macrophages.
Jing Wang +9 more
wiley +1 more source
Studying the Serum as Well as Serous Level of IL-17 and IL-23 in Patients with Serous Otitis Media
Iranian Journal of Allergy, Asthma and Immunology, 2017 Serous otitis media with effusion (OME) is a middle ear inflammatory response to allergens and microbes which stimulate leukocytes to produce different inflammatory mediators after obstruction of Eustachian tube. Here, we investigated the levels of these
Ahmad Yeghaneh Moghaddam +3 more
doaj
Targeting Interleukin-17 as a Novel Treatment Option for Fibrotic Diseases. [PDF]
J Clin Med, 2023 Sisto M, Lisi S.
europepmc +1 more source
CpG Oligodeoxynucleotide Protection in Polymicrobial Sepsis Is Dependent on Interleukin‐17 [PDF]
, 2005 Lauren Rice +7 more
openalex +1 more source
Inorganic Nanomaterials Meet the Immune System: An Intricate Balance
Advanced Healthcare Materials, Volume 14, Issue 11, April 25, 2025.Metal nanomaterials influence the immune responses, such as cytokine production and T‐cell activity. Unintended immune modulation can have significant consequences while its fine‐tuning may transform the oncology care. Within this review, a critical analysis is offered regarding the nano/immune‐interaction and suggests the future research directions to
Gloria Pizzoli +3 more
wiley +1 more source
Interleukin-17 as a key player in neuroimmunometabolism. [PDF]
Nat Metab, 2023 Douglas A, Stevens B, Lynch L.
europepmc +1 more source
Advanced Healthcare Materials, Volume 14, Issue 13, May 16, 2025.Short‐chain fatty acids modulate anti‐ROR1 chimeric antigen receptor (CAR) T‐cell function in an intestinal adenocarcinoma‐on‐chip model. Butyrate and propionate reduce cytotoxicity, cytokine release, and infiltration by inducing a regulatory T‐cell phenotype and T‐cell exhaustion via histone deacetylase inhibition.
Valentin D. Wegner +5 more
wiley +1 more source