Results 261 to 270 of about 469,171 (282)
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<p>Effects of Remote Ischemic Conditioning on Cerebral Hemodynamics in Ischemic Stroke</p>
Neuropsychiatric Disease and Treatment, 2020Yihe Zhang, Chen Qin, Hang Jin
exaly
The Role of Platelets in Ischemic Conditioning.
Conditioning medicineIschemic heart disease (IHD) is one of the leading causes of death and disability worldwide. Platelets, as the main regulators of hemostasis, are major players in acute myocardial ischemia/reperfusion injury (IRI). Additionally, platelets are modified by endogenous cardioprotective strategies such as ischemic preconditioning, postconditioning, and ...
Juan, Alpuche +5 more
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RECAST (Remote Ischemic Conditioning After Stroke Trial)
Stroke, 2017Timothy J England +2 more
exaly
[Therapeutic perspectives in ischemic conditions].
Recenti progressi in medicina, 2005Recently the angiogenic mechanism of platelet-derived growth factor-CC was discovered. This factor is capable of mobilize endothelial progenitor cells in ischemic conditions, and induce differentiation of bone marrow cells into endothelial cells. The possibility of modulating the activity of this factor may provide a new strategy for treating ischemic ...
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Chemical Conditioning as an Approach to Ischemic Stroke Tolerance: Mitochondria as the Target
International Journal of Molecular Sciences, 2016Liang-Jun Yan, Zhen Jin, Jinzi Wu
exaly
The Angiogenic Effects of Ischemic Conditioning in Experimental Critical Limb Ischemia
European Journal of Vascular and Endovascular Surgery, 2014Murat Alper
exaly
The Role of Heme Oxygenase-1 in Remote Ischemic and Anesthetic Organ Conditioning
Antioxidants, 2019Inge Bauer, Raupach Annika
exaly
Effect of long-term remote ischemic conditioning in patients with chronic ischemic heart failure
Basic Research in Cardiology, 2017Steffen Ringgaard +2 more
exaly
THE ROLE OF THE SVZ IN ISCHEMIC CONDITION
2012The occlusion of a cerebral artery or stroke often results in neuronal deficit and/or patient death. A partial recovery often follows non-fatal stroke and this may be due to the activation of the progenitor cells in the Sub- Ventricular Zone (SVZ) naturally occurring after ischemia.
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