Results 41 to 50 of about 2,143,132 (361)

Effect of pesticides “Butylcaptax (Russia)” and “Droppa (Russia)” on respiration and oxidative phosphorylation of liver mitochondria of pregnant rats and their embryos [PDF]

open access: yesE3S Web of Conferences, 2021
The article provides information on the effect of pesticides - butylcaptax and droppa on respiration and oxidative phosphorylation of liver mitochondria in rat rats and their embryos.
Alimova Rano   +2 more
doaj   +1 more source

Mitochondria-targeted antioxidant mitoquinone attenuates liver inflammation and fibrosis in cirrhotic rats.

open access: yesAmerican Journal of Physiology - Gastrointestinal and Liver Physiology, 2019
BACKGROUND AND AIMS In liver cirrhosis, oxidative stress plays a major role in promoting liver inflammation and fibrosis. Mitochondria dysregulation is responsible for excessive reactive oxygen species production.
S. Turkseven   +5 more
semanticscholar   +1 more source

Spermine binding to liver mitochondria

open access: yesBiochimica et Biophysica Acta (BBA) - Biomembranes, 1996
Non-equilibrium binding of spermine to mitochondrial membranes is studied in rat liver mitochondria by applying a new thermodynamic treatment of ligand-receptor interactions (Di Noto, V., Dalla Via, L., Toninello, A. and Vidali, M. (1996) Macromol. Theory Simul. 5, 165-181).
DALLA VIA, LISA   +3 more
openaire   +3 more sources

Inhibitory Effects of Resveratrol against Mitochondrial Toxicity Induced by Perfluorooctanoic Acid in Isolated Liver Mitochondria of Male Rats

open access: yesJournal of Mazandaran University of Medical Sciences, 2022
Background and purpose: Resveratrol (RSV) is a naturally polyphenolic compound found in grapes and other plant sources. It has potent free radical scavenger and antioxidative properties.The aim of this study was to evaluate the protective effect of ...
Maloos Naderi   +3 more
doaj  

The Role of l-Carnitine in Mitochondria, Prevention of Metabolic Inflexibility and Disease Initiation

open access: yesInternational Journal of Molecular Sciences, 2022
Mitochondria control cellular fate by various mechanisms and are key drivers of cellular metabolism. Although the main function of mitochondria is energy production, they are also involved in cellular detoxification, cellular stabilization, as well as ...
M. Virmani, Maria Cirulli
semanticscholar   +1 more source

Succinate oxidation by human liver mitochondria [PDF]

open access: yesArchives of Biochemistry and Biophysics, 1965
Factors controlling the rate of oxidation of succinate were studied in mitochondria isolated from twenty-seven specimens of normal human liver obtained at operation. These were compared with mitochondria prepared from the livers of anesthetized and unanesthetized rats.
Henley, Keith S., Laughrey, Earl G.
openaire   +3 more sources

Adaptations of hepatic lipid metabolism and mitochondria in dairy cows with mild fatty liver.

open access: yesJournal of Dairy Science, 2018
The inevitable deficiency in nutrients and energy at the onset of lactation requires an optimal adaptation of the hepatic metabolism to overcome metabolic stress. Fatty liver is one of the main health disorders after parturition.
Xiliang Du   +15 more
semanticscholar   +1 more source

Biogenesis of mitochondrial porin [PDF]

open access: yes, 1990
We review here the present knowledge about the pathway of import and assembly of porin into mitochondria and compare it to those of other mitochondrial proteins.
A. Grossmann   +74 more
core   +1 more source

Phosphorylation of purine and pyrimidine nucleosides by isolated rat liver mitochondria [PDF]

open access: yes, 1975
Formation of 5'-AMP, 5'-GMP, 5'-CMP and 5'UMP was confirmed in isolated rat liver mitochondria incubated with alpha-ketoglutarate, inorganic phosphate, purine nucleoside and pyrimidine nucleoside. Increased incorporation of 32Pi into ATP, GTP and UTP was
Inaba, Kozo, Oda, Takuzo
core   +1 more source

Antioxidants that protect mitochondria reduce interleukin-6 and oxidative stress, improve mitochondrial function, and reduce biochemical markers of organ dysfunction in a rat model of acute sepsis [PDF]

open access: yes, 2013
Funding This study was funded by the Medical Research Council (Grant number G0800149). Research material from this study is not available. Acknowledgement We are very grateful to Dr Robin A.J. Smith, Department of Chemistry, University of Otago, Dunedin,
Galley, H. F.   +3 more
core   +1 more source

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