Results 251 to 260 of about 76,910 (294)

Slc44a2 Deficiency Unveils an IFN‐I–Dependent Feedback Control of pDC Egress

Advanced Science, Volume 13, Issue 53, 24 September 2026.
Working model of SLC44A2‐mediated maintenance of pDC homeostasis. This model illustrates two central mechanisms by which SLC44A2 regulates pDC homeostasis: (1) SLC44A2 limits IFN‐I production by exporting amino acids (T, N, Q), thereby preventing spontaneous pDC activation.
Ruiqun Chen, Tao Wu, Zhen Shi, Ravi Kumar Verma, Linlin Sheng, Lei Tao, Mingming Yang, Yuchen Wang, Conggang Zhang, Hao Fan, Li Wu, Ligong Chen   +11 more
wiley   +1 more source

Toll-like receptor 9-overexpressing red blood cell biomimetic microbubbles adsorb cell-free DNA to relieve steatotic liver ischemia-reperfusion injury. [PDF]

Mater Today Bio
Du J, Liu J, Chen S, Deng H, Ye H, Xiao X, Yang J, Yang J, Zhang Z, Yin T, Yao W, Hei Z.   +11 more
europepmc   +1 more source

Mogroside V Alleviates Renal Injury in Diabetic Mice via Regulation of the <i>TLR4</i>/<i>NF-κB</i> Pathway and Modulation of ECM Remodeling. [PDF]

Int J Mol Sci
Guo X, Shao H, Zhang J, Xie X, Peng D, Xu Q.   +5 more
europepmc   +1 more source

Beyond receptor activation: biased toll-like receptor signaling in periodontal inflammation and regeneration. [PDF]

Front Immunol
Mekhemar M, Hassanein FEA, Abou-Bakr A.
europepmc   +1 more source

Quantitative MYD88 L265P and flow cytometry levels for outcome determination in IgM gammopathies: the SAL-TO study. [PDF]

Blood Adv
Dogliotti I, Jiménez C, Peri V, Ferrante M, Musto D, Mercadante S, Zaccaria GM, Ghislieri M, Benevolo G, Ocio EM, Rubio A, Murillo I, Escalante F, Aguilera C, García MA, García de Coca A, Hernández R, Dávila J, Cavallo F, Puig N, González de la Calle V, Sarasquete ME, Alcoceba M, Ragaini S, Clerico M, Consoli C, Amaducci E, García-Álvarez M, Chillón MDC, Medina A, Díaz MG, Gutiérrez NC, Bruno B, Drandi D, Ferrero S, García-Sanz R.   +35 more
europepmc   +1 more source

10,11-Dehydrocurvularin attenuates LPS-induced neuroinflammation in BV2 cells by inhibiting the TLR2/MyD88/NLRP3 signaling pathway. [PDF]

Biochem Biophys Rep
Tian C, Sun J, Wang J, Wang Y, Wan P, Sun G, Mu Y, Geng M, Xu B, Zhang Y.   +9 more
europepmc   +1 more source

MYD88/TRIF Signaling, Pluripotency and Klotho Regulation in the Intestine, Kidneys, Liver, and Lungs of a Septic Mouse Model. [PDF]

Curr Issues Mol Biol
Erodotou M, Kapelouzou A, Mylonas KS, Soukouli I, Boletis JN, Tsourouflis G, Liakakos T, Schizas D.   +7 more
europepmc   +1 more source

Oncogenic MyD88

Science Signaling, 2011
A gain-of-function mutation in MyD88 promotes survival of a B cell lymphoma through activation of NF-κB and JAK-STAT signaling pathways.
John F. Foley
openaire   +2 more sources

Some of the next articles are maybe not open access.

Related searches:

Mycobacterial Infection in MyD88‐Deficient Mice

Microbiology and Immunology, 2003
AbstractMyD88 is an adaptor protein that plays a major role in TLR/IL‐1 receptor family signaling. To understand the role of MyD88 in the development of murine tuberculosis in vivo, MyD88 knockout (KO) mice aerially were infected with Mycobacterium tuberculosis. Infected MyD88 mice were not highly susceptible to M.
Shizuo Akira, Hiroyuki Yamada
exaly   +3 more sources

Lasalocid A selectively induces the degradation of MYD88 in lymphomas harboring the MYD88 L265P mutation

Blood
Abstract Myeloid differentiation primary response protein 88 (MYD88) is a key adaptor molecule in the signaling pathways of toll-like receptor and interleukin-1 receptor. A somatic mutation resulting in a leucine-to-proline change at position 265 of the MYD88 protein (MYD88 L265P) is one of the most prevalent ...
Wei Li, Ruirui Wang, Junhao Wang, Dafei Chai, Xiaohui Xie, Ken H. Young, Ya Cao, Yong Li, Xinfang Yu   +8 more
openaire   +3 more sources