Results 121 to 130 of about 1,083 (144)

Alpha-actinin-1, profilin-1, filamin-A, integrin alpha-M and glutathione synthase were identified to be associated with cognitive and social functions of schizophrenia. [PDF]

Sci Rep
Fu X, Fang X, Yang L, Liu Y, Jia F, Ye G, Yuan L, Zhang X, Liu Y, Li C, Du X.   +10 more
europepmc   +1 more source

Clinical impact of ceruloplasmin levels at ANCA-associated vasculitis diagnosis. [PDF]

PLoS One
Camboulive L, Grandhomme F, Martin Silva N, Khoy K, Mariotte D, Lobbedez T, Dumont A, Nguyen A, de Boysson H, Aouba A, Deshayes S.   +10 more
europepmc   +1 more source

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Truncation of Sp1 transcription factor by myeloblastin in undifferentiated HL60 cells

Journal of Cellular Physiology, 1998
When HL60 cells are exposed to 1,25-dihydroxyvitamin D3 (1,25D3), they undergo changes approximating the phenotype of the monocyte. Little is known, however, about the regulation and the mechanisms of this transition. It was previously noted that DNA binding by the Sp1 transcription factor in nuclear extracts of HL60 cells is profoundly altered when ...
Jia Rao, Neil L Spector, George P Studziński   +2 more
exaly   +3 more sources

Human Myeloblastin (Leukocyte Proteinase 3): Reactions with Substrates, Inactivators and Activators in Comparison with Leukocyte Elastase

Biological Chemistry Hoppe-Seyler, 1996
Human myeloblastin (leukocyte proteinase 3) shares many biochemical properties with leukocyte elastase, but rapidly loses enzymatic activity when raising the pH and/or the ionic strength of an acidic solution or when handled in glass vessels. This poses limits to kinetic experiments requiring long incubation times.
B A Michel, Antonio Baici
exaly   +3 more sources

Myeloblastin is an Myb target gene: mechanisms of regulation in myeloid leukemia cells growth-arrested by retinoic acid

Blood, 2001
AbstractA pivotal role has been assigned to Myb in the control of myeloid cell growth. Although Myb is a target of retinoic acid, little is known about the mechanisms by which it may contribute to induced growth arrest in leukemia cells. Indeed, few Myb target genes are known to be linked to proliferation.
Pierre G Lutz, Christel Moog-Lutz
exaly   +4 more sources

Myeloblastin/Proteinase 3 Belongs to the Set of Negatively Regulated Primary Response Genes Expressed During in Vitro Myeloid Differentiation

Biochemical and Biophysical Research Communications, 1994
Regulation of myeloblastin expression was studied during myeloid differentiation of HL60 cells and compared with that of myeloperoxidase. Induction towards the monocytic pathway of differentiation caused a rapid, marked and parallel down-regulation of both transcripts which were insensitive to cycloheximide. Thus, myeloblastin is directly regulated and,
A Ziemiecki, M F Fey, A Tobler
exaly   +4 more sources

A myeloblastin/proteinase-3 cDNA clone identifies a BglII and a PvuII restriction fragment length polymorphism

Human Genetics, 1993
A myeloblastin/proteinase-3 (MBN/PR-3) cDNA probe detects two bi-allelic (BglII, PvuII) DNA polymorphisms. These restriction fragment length polymorphisms provide new genetic markers on chromosome 19.
M F Fey, A Tobler
exaly   +4 more sources

The Phylogeny of Proteinase 3/Myeloblastin, the Autoantigen in Wegener's Granulomatosis, and Myeloperoxidase as Shown by Immunohistochemical Studies on Human Leukemic Cell Lines

Clinical Immunology and Immunopathology, 1994
Proteinase 3/myeloblastin (Pr3/MBN) is a serine protease found in primary granules of neutrophilic granulocytes and monocytes in man. This enzyme has been identified as the main autoantigen in Wegener's granulomatosis. Pr3/MBN was earlier identified in the promyelocytic cell line HL-60 and was assumed to be at least partly responsible for controlling ...
J Minowada, M Lanotte, A Wiik
exaly   +3 more sources

Interferon-α, but not the ABL-kinase inhibitor imatinib (STI571), induces expression of myeloblastin and a specific T-cell response in chronic myeloid leukemia

Blood, 2003
AbstractChronic myeloid leukemia (CML) is a clonal disease of hematopoietic stem cells caused by a reciprocal translocation of the long arms of chromosomes 9 and 22. In human leukocyte antigen A*0201+ (HLA-A*0201+) individuals, response after interferon-α (IFN-α) was shown to be associated with the emergence of CML-specific cytotoxic T cells that ...
Andreas Burchert, Cornelia Brendel, Dali Cai   +2 more
exaly   +3 more sources

[3] Myeloblastin: Leukocyte proteinase 3

Methods in Enzymology, 1994
John R Hoidal, N V Rao
exaly   +2 more sources