Results 131 to 140 of about 44,548 (260)
Phenotypical differences and thrombosis rates in secondary erythrocytosis versus polycythemia vera
Eliane Nguyen +8 more
doaj +1 more source
Neutralize the neutrophils! Neutrophil β1 /β2 integrin activation contributes to JAK2-V617F–driven thrombosis [PDF]
Barbui +4 more
core +2 more sources
This Is Not a Myeloproliferative Neoplasm…
Pediatric Blood &Cancer, Volume 73, Issue 7, July 2026.
Stephanie Juané Kennedy
wiley +1 more source
Abstract Background Mutations in cytokine receptor and JAK/STAT; Cy‐JAK/STAT) signaling genes drive myeloproliferative neoplasms (MPNs) but remain incompletely characterized in acute myeloid leukemia (AML). The authors evaluated the prevalence, clinical presentation, and prognostic significance of Cy‐JAK/STAT pathway mutations in patients with AML and ...
Moath Albliwi +22 more
wiley +1 more source
American Journal of Hematology, Volume 101, Issue 6, Page 1239-1243, June 2026.
Michelle Pirotte +6 more
wiley +1 more source
Associació de l'estat mutacional de JAK2 i les anomalies citogenètiques en les neoplàsies mieloproliferatives cròniques Filadèlfia negatives : Correlació amb les dades clíniques i analítiques [PDF]
L'estudi correlaciona l'estat mutacional de JAK2V617F amb les alteracions citogenètiques, algunes dades clíniques i paràmetres de laboratori i valorar l'impacte sobre la progressió i pronòstic de la malaltia de 526 casos de NMPc Ph-.
Feliu Frasnedo, Evarist +4 more
core
MESENCHYMAL STEM CELLS IN PH-NEGATIVE MYELOPROLIFERATIVE NEOPLASMS: IDLE OBSERVERS OR PART OF THE DISEASE? [PDF]
Accurso, V. +8 more
core +1 more source
ABSTRACT Background Janus kinase inhibitors (JAKis), the current standard of care for myelofibrosis (MF), provide clinical benefit, but responses are frequently incomplete, non‐durable, and associated with cytopenias, underscoring the need for therapies with novel mechanisms of action.
Sebastian Grosicki +15 more
wiley +1 more source

