Results 191 to 200 of about 12,605,538 (213)

Effect of Neurokinin-1 Receptor Antagonists on Experimental Postoperative Adhesion: A Systematic Review and Meta-Analysis with Trial Sequential Analysis of Preclinical Studies. [PDF]

Medicina (Kaunas)
Hwang HJ, Choi GJ, Choi YS, Kim BG, Kang H.   +4 more
europepmc   +1 more source

Interactions Between Neurokinin B and Kisspeptin in Mediating Estrogen Feedback in Healthy Women.

J Clin Endocrinol Metab, 2016
Skorupskaite K, George JT, Veldhuis JD, Millar RP, Anderson RA.   +4 more
europepmc   +1 more source

Modeling human neurokinin-1 receptor structure using the crystal structure of bovine rhodopsin [PDF]

, 2005
Coutinho, Evans C., Khedkar, Santosh A., Malde, Alpeshkumar K.   +2 more
core  

Genetic, neuropeptidergic, and cardiometabolic interplay in female central precocious puberty. [PDF]

Cardiovasc Endocrinol Metab
Musayeva AK, Amirova MF.
europepmc   +1 more source

A Comprehensive Method To Quantify Adaptations by Male and Female Mice With Hot Flashes Induced by the Neurokinin B Receptor Agonist Senktide.

Endocrinology, 2017
Krull AA, Larsen SA, Clifton DK, Neal-Perry G, Steiner RA.   +4 more
europepmc   +1 more source

Advances in Pharmacotherapy for Menopausal Vasomotor Symptoms. [PDF]

Drugs
Young Moss S, Lee A, Simon JA.
europepmc   +1 more source

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Serum kisspeptin, neurokinin B and inhibin B levels can be used as alternative parameters to distinguish idiopathic CPP from premature thelarche in the early stages of puberty

Clinical Endocrinology, 2023
There is controversial results about serum kisspeptin, neurokinin‐B (NKB), anti‐Müllerian hormone (AMH) and inhibin B (INHB) levels in girls with central precocious puberty (CPP).
D. Vurallı, Nurdan Çiftçi, H. Demirbilek   +2 more
semanticscholar   +1 more source

Serum kisspeptin, leptin, neuropeptide Y, and neurokinin B levels in adolescents with polycystic ovary syndrome

Journal of Pediatric Endocrinology & Metabolism (JPEM), 2022
Objectives Polycystic ovary syndrome (PCOS) is characterized by ovarian dysfunction, clinical and/or biochemical hyperandrogenism, and polycystic ovaries. Its pathogenesis is still unclear.
I. Guzelkas, Z. Orbak, H. Doneray, Nurinnisa Ozturk, N. Sagsoz   +4 more
semanticscholar   +1 more source