Results 191 to 200 of about 484,958 (335)
Invariant Aspartic Acid in Muscle Nicotinic Receptor Contributes Selectively to the Kinetics of Agonist Binding [PDF]
Won Yong Lee, Steven M. Sine
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Effects of an Alpha 7-Nicotinic Agonist on Default Network Activity in Schizophrenia
J. Tregellas+10 more
semanticscholar +1 more source
ABSTRACT Introduction ADHD is often associated with comorbid psychiatric conditions. Differential diagnosis between other conditions and ADHD is not always clear, and patients are sometimes initially treated for another disorder instead. ADHD diagnosis and appropriate ADHD treatment potentially reduce the need for medication of the other disorder ...
Elisa Westman+8 more
wiley +1 more source
Ex vivo receptor occupation of GTS-21, a novel nicotinic receptor agonist, in rats.
Yoshihisa Nozawa+4 more
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The characterization of a novel rigid nicotine analog with α7-selective nAChR agonist activity and modulation of agonist properties by boron inclusion [PDF]
Roger L. Papke+4 more
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Abstract Aims This cross‐sectional study examined associations between self‐reported taste perception changes and appetite‐related outcomes in individuals with obesity treated with glucagon‐like peptide‐1 receptor agonist (GLP‐1 RAS) or dual glucose‐dependent insulinotropic polypeptide (GIP)/GLP‐1 RAS in real‐world conditions.
Ali Kapan+4 more
wiley +1 more source
Modulation of airway inflammation and resistance in mice by a nicotinic receptor agonist [PDF]
M-R. Blanchet+2 more
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Functional Magnetic Resonance Imaging of Effects of a Nicotinic Agonist in Schizophrenia
J. Tregellas+10 more
semanticscholar +1 more source
Pischiutta F and coworkers identify prostaglandins and kynurenine as key mediators of the mesenchymal stromal cells‐derived secretome's neuroprotective effects. A synthetic cocktail composed of these factors promotes long‐term functional recovery, reduces brain structural damage, and modulates neuroinflammation after traumatic brain injury, supporting ...
Francesca Pischiutta+24 more
wiley +1 more source
Physics of Protein Aggregation in Normal and Accelerated Brain Aging
Soluble monomeric proteins precipitate via nucleation into insoluble amyloids in response to age‐related exposures (e.g., microbes, nanoparticles). Persistent soluble‐to‐insoluble phase transition depletes the functional proteins. In normal aging, replacement matches loss; in accelerated aging, it does not.
Alberto J. Espay+9 more
wiley +1 more source