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Plasminogen activator inhibitor-1 and the kidney
American Journal of Physiology-Renal Physiology, 2002Plasminogen activator inhibitor-1 (PAI-1) is a serine protease inhibitor that was isolated 20 years ago. First recognized as an inhibitor of intravascular fibrinolysis, it is now evident that PAI-1 is a multifunctional protein with actions that may be dependent on or independent of its protease inhibitory effects.
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Plasminogen activator inhibitor 1 in thrombotic disease
Current Opinion in Hematology, 1996Impaired fibrinolytic function, mainly due to an elevation of plasma plasminogen activator inhibitor 1 concentration, is a common finding in patients with thrombotic disease. In patients subjected to hip surgery, preoperatively increased levels of plasminogen activator inhibitor 1 seem to be predictive of postoperative deep venous thrombosis.
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Plasminogen activator inhibitor 1 in human carcinoma tissues
International Journal of Cancer, 1991AbstractThe content of PAI‐1 was measured in carcinoma tissues from the stomach and colorectum divided macroscopically into 3 portions: the central part of the carcinoma, the marginal part of the carcinoma containing some normal mucosa, and the normal mucosa. Among these tissues, the highest levels of PAI‐1 antigen were found in the central part of the
N, Tanaka +5 more
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Plasminogen Activator Inhibitor 1 and Atherothrombosis
Thrombosis and Haemostasis, 1993I, Juhan-Vague, M C, Alessi
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Recombinant Plasminogen Activator Inhibitor-1
Drugs of the Future, 1992T.M. Reilly, A.L. Racanelli, S.A. Mousa
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P55. Plasminogen activator inhibitor-1
Blood Coagulation & Fibrinolysis, 1996R. W. Faint +8 more
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Vascular Regulation of Plasminogen Activator Inhibitor-1 Activity
Seminars in Thrombosis and Hemostasis, 1994J M, Edelberg, D C, Sane, S V, Pizzo
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Update on plasminogen activator inhibitor-1.
The Canadian journal of cardiology, 1998Plasminogen activator inhibitor-1 (PAI-1) is produced by the endothelium and determines the rate and extent of fibrinolysis. Recent data on PAI-1 gene polymorphism and the potential role of glucose in regulating PAI-1 expression in vascular disease are reviewed.
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