Results 261 to 270 of about 157,697 (307)
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Protonation-deprotonation of purines and purine nucleosides
Journal of Molecular Structure: THEOCHEM, 1989Abstract The protonation of purines and purine nucleosides at N7 and the HC8 acidity of the cations have been explored by semi-empirical and ab initio MO ...
Donald W. Boerth, Pradip K. Bhowmik
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New England Journal of Medicine, 1970
"The biochemistry of uric acid and its relation to gout" was recently (1963) reviewed in a series of articles that appeared in the Medical Progress section of the Journal.1 In this series the bioch...
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"The biochemistry of uric acid and its relation to gout" was recently (1963) reviewed in a series of articles that appeared in the Medical Progress section of the Journal.1 In this series the bioch...
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Biochemical Society Transactions, 1981
Abstract The inherited disorders of purine metabolism associated with renal disease are listed in Table 24.1. They are the sole cause of only a small proportion of all renal disease and are therefore best considered as part of the overall problem of purine, especially uric acid metabolism-related renal disease.
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Abstract The inherited disorders of purine metabolism associated with renal disease are listed in Table 24.1. They are the sole cause of only a small proportion of all renal disease and are therefore best considered as part of the overall problem of purine, especially uric acid metabolism-related renal disease.
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3 Purine, Purine Nucleoside, and Purine Nucleotide Aminohydrolases
1971Publisher Summary This chapter discusses the chemical significance of adenosine aminohydrolase, 5′-adenylic acid aminohydrolase, adenine nucleoside, nucleotide aminohydrolase, guanine aminohydrolase, and guanosine aminohydrolase. The partially purified adenine aminohydrolase from Azotobacter vinelandii catalyzes the anaerobic conversion of adenine ...
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2003
The activation of adenosine A1, A2 andA3 receptors can protect neurones against damage generated by mechanical or hypoxic/ischaemic insults as well as excitotoxins. A1 receptors are probably effective by suppressing transmitter release and producing neuronal hyperpolarisation.
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The activation of adenosine A1, A2 andA3 receptors can protect neurones against damage generated by mechanical or hypoxic/ischaemic insults as well as excitotoxins. A1 receptors are probably effective by suppressing transmitter release and producing neuronal hyperpolarisation.
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Journal of Cellular and Comparative Physiology, 1951
GAR transformylase, which is responsible for the incorporation of formate carbon into what becomes position 8 of the purine ring, has been completely separated from 5-amino-4-imidazolecarboxamide ribotide transformylase (3). The latter enzyme is responsible for the incorporation of formate carbon into position 2 of the purine ring.
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GAR transformylase, which is responsible for the incorporation of formate carbon into what becomes position 8 of the purine ring, has been completely separated from 5-amino-4-imidazolecarboxamide ribotide transformylase (3). The latter enzyme is responsible for the incorporation of formate carbon into position 2 of the purine ring.
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Life Sciences, 1987
The fact that purine nucleotides and nucleosides are so important in normal cellular functions makes their analysis complicated. In order to measure tissue levels, samples have to be very rapidly fixed. Measurement in blood is complicated by the risk of releasing purine nucleotides during the sampling procedure and by the possibility of very rapid ...
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The fact that purine nucleotides and nucleosides are so important in normal cellular functions makes their analysis complicated. In order to measure tissue levels, samples have to be very rapidly fixed. Measurement in blood is complicated by the risk of releasing purine nucleotides during the sampling procedure and by the possibility of very rapid ...
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Inhibition of Lipid Peroxidation by Purines and Analogues of Purine
1995The importance of oxygen radicals in tissue injury is widely recognized(1). Mutation, cancer, inflammation, and ageing are thought to be caused by oxygen radicals. It has also been confirmed that lipids are peroxidized by oxygen radicals leading to the damaging effects on cellular constituents. Mickel & Horbar(2) reported that peroxidized lipids affect
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