Results 181 to 190 of about 23,457 (205)
ATPase copper transporting beta contributes to cisplatin resistance as a regulatory factor of extracellular vesicles in head and neck squamous cell carcinoma. [PDF]
Cancer Gene TherOgawa T +13 more
europepmc +1 more source
Correction to "From Mitochondria to Immunity: The Emerging Roles of Mitochondria-Derived Vesicles and Small Extracellular Vesicles in Cellular Communication and Disease". [PDF]
J Extracell Vesicleseuropepmc +1 more source
Fluid shear stress-mediated upregulated exosomal-IGF2 promotes hepatocellular carcinoma cell migration and metastasis. [PDF]
Am J Cancer ResFeng T +9 more
europepmc +1 more source
SNX16 aggravates AngII-induced cardiac hypertrophy in mice via EGFR transactivation. [PDF]
Commun BiolXie L +17 more
europepmc +1 more source
Some of the next articles are maybe not open access.
Related searches:
Related searches:
Potential role of Acanthamoeba Rab7
Experimental Parasitology, 2022Acanthamoeba castellanii is a free-living protozoan that causes several severe human parasitic diseases such as Acanthamoeba keratitis and granulomatous encephalitis. A. castellanii feeds on bacteria, yeasts, and other organic particles as food sources, but the mechanisms of digestion in acanthamoebal cells are unclear.
Zih-Bin, Hong +3 more
openaire +2 more sources
Biochemical Society Transactions, 2009
The CMT2B (Charcot–Marie–Tooth type 2B) disease is an autosomal dominant axonal neuropathy. Sensory loss, distal muscle weakness and wasting, frequent foot ulcers and amputations of the toes due to frequent infections characterize this neuropathy. Four missense mutations in the rab7 gene have been identified as causative of the disease. Rab7 is a small
Laura Cogli +2 more
openaire +4 more sources
The CMT2B (Charcot–Marie–Tooth type 2B) disease is an autosomal dominant axonal neuropathy. Sensory loss, distal muscle weakness and wasting, frequent foot ulcers and amputations of the toes due to frequent infections characterize this neuropathy. Four missense mutations in the rab7 gene have been identified as causative of the disease. Rab7 is a small
Laura Cogli +2 more
openaire +4 more sources
1995
Abstract Rab7 was first identified by screening a cDNA library from BRL-3A cells (buffalo rat liver) and initially called BRL-ras (Bucci et al. 1988). Later, a cDNA library from MDCK (Madin-Darby canine kidney) cells was screened with degenerated oligonucleotides made in the conserved domains and the canine clone was isolated (Chavrier ...
Cecilia Bucci +2 more
openaire +3 more sources
Abstract Rab7 was first identified by screening a cDNA library from BRL-3A cells (buffalo rat liver) and initially called BRL-ras (Bucci et al. 1988). Later, a cDNA library from MDCK (Madin-Darby canine kidney) cells was screened with degenerated oligonucleotides made in the conserved domains and the canine clone was isolated (Chavrier ...
Cecilia Bucci +2 more
openaire +3 more sources
Genetic mapping of the mouse Rab7 gene and pseudogene and of the human RAB7 homolog
Mammalian Genome, 1998Rab proteins are small GTP-ases localized to distinct membrane compartments in eukaryotic cells and regulating specific steps of intracellular vesicular membrane traffic. The Rab7 protein is localized to the late endosomal compartment and controls late steps of endocytosis.
CHIARIELLO M +6 more
openaire +5 more sources