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NSUN5 Attenuates Renal Injury and Ferroptosis in Hyperuricaemic Nephropathy Through YBX2‐Dependent Stabilisation of SCD1 m5C Methylation

Advanced Science, EarlyView.
NSUN5 is downregulated in hyperuricaemic nephropathy. Overexpression of NSUN5 enhances the stability of SCD1 mRNA through the m5C reader YBX2, ultimately inhibiting ferroptosis in renal tubular epithelial cells. Additionally, this axis suppresses the NF‐κB signaling pathway to alleviate inflammation and upregulates ABCG2 to promote uric acid excretion,
Xiu‐xiu Song, Xiao‐guo Suo, Yue Yu, Kuo Zhang, Chen‐ao Li, Jie Wang, Hui‐xia Xu, Si‐yu Niu, Dong‐xue Lv, Zi‐hao He, Feng‐he Li, Xiao‐ming Meng, Juan Jin   +12 more
wiley   +1 more source

Damnacanthal Suppresses Breast Cancer Cells by Inducing Apoptosis and Cell Cycle Arrest via NF-ĸB Signaling. [PDF]

Pharmacol Res Perspect
Jongcharoen O, Reabroi S, Sanvarinda P, Tungmunnithum D, Parichatikanond W, Pinthong D.   +5 more
europepmc   +1 more source

Age- and Genotype-Associated Specific Expression of IL-1 and TNF Receptors on Immunocompetent Cells. [PDF]

Int J Mol Sci
Zhukova J, Lopatnikova J, Vasilyev F, Alshevskaya A, Lipa D, Sennikov S.   +5 more
europepmc   +1 more source

Regulatory B cells in the central nervous system: From immune regulation to neuroprotection. [PDF]

Neuroprotection
Stanaszek L, Janowski M.
europepmc   +1 more source

Research progress on the intrinsic link between the occurrence and progression of malignant tumors, chronic kidney disease and drug safety. [PDF]

Am J Cancer Res
Zhang D, Jiang S, Liu L, Lu J, Li W.
europepmc   +1 more source

Hepatocellular carcinoma's inflammatory secret: ferroptosis as the key. [PDF]

Transl Cancer Res
Caro AA, LoBianco FV, Aykin-Burns N.
europepmc   +1 more source

Obesity and cancer: Relevance of DNA damage response. [PDF]

Transl Oncol
Deshmukh B, Ajay AK, Bhat MK.
europepmc   +1 more source

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The Tumor Necrosis Factor Ligand and Receptor Families

New England Journal of Medicine, 1996
Tumor necrosis factor (TNF) and lymphotoxin-α were isolated more than 10 years ago, on the basis of their ability to kill tumor cells in vitro and to cause hemorrhagic necrosis of transplantable tumors in mice.1 The complementary DNAs and genes encoding each protein were cloned immediately thereafter.2,3 Concurrently, a factor known as cachectin was ...
BAZZONI, Flavia, BEUTLER B.
openaire   +5 more sources