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Curcumin-Piperine Self-Nanoemulsifying Delivery in <i>Zanthoxylum rhetsa</i> Seed Oil Attenuates Cuprizone-Induced Frontal Cortex Toxicity. [PDF]
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REMYELINATION IN MULTIPLE SCLEROSIS
Journal of Neuropathology and Experimental Neurology, 1978AbstractChronic plaques in central nervous system tissue fixed by in situe perfusion for electron microscopy were examined for evidence of remyelination in 2 patients with multiple sclerosis (MS). Fibers with abnormal central myelin sheaths of several types were found at the margins of most of the plaques studied. The most common of these were: (1) the
J W, Prineas, F, Connell
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NeuroReport, 2002
Remyelination of demyelinated axons in the CNS is a regenerative process that, like many others, becomes less efficient with age. This article reviews a series of studies in which toxin models of demyelination have been used to characterize this phenomenon.
Robin J M, Franklin +2 more
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Remyelination of demyelinated axons in the CNS is a regenerative process that, like many others, becomes less efficient with age. This article reviews a series of studies in which toxin models of demyelination have been used to characterize this phenomenon.
Robin J M, Franklin +2 more
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Macrophages and CNS remyelination
Journal of Neurochemistry, 2014AbstractMicroglia are the resident macrophages of the central nervous system that survey the microenvironment for signals of injury or infection. The response to such signals induces an inflammatory response involving macrophages derived from both resident microglia and recruited circulating monocytes.
Veronique E, Miron, Robin J M, Franklin
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TREM2 activation promotes remyelination
Nature Reviews Neurology, 2020Cignarella, F. et al. TREM2 activation on microglia promotes myelin debris clearance and remyelination in a model of multiple sclerosis. Acta Neuropathol. https://doi.org/10.1007/s00401-020-02193-z (2020) Article PubMed Google Scholar Ulland, T. K. & Colonna, M. TREM2 — a key player in microglial biology and Alzheimer disease. Nat. Rev. Neurol. 14, 667–
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Remyelination in multiple sclerosis
Multiple Sclerosis Journal, 1997Stimulation of remyelination in multiple sclerosis lesions may be one possible therapeutic approach. However, since the pathology and pathogenesis of multiple sclerosis may be heterogeneous, it is important to analyze in what patients and at what stages of the disease such a therapy can be reasonable and feasible.
H, Lassmann +3 more
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Multiple Sclerosis Journal, 1997
Now that the concept of remyelination is gaining acceptance in MS, there is a need for a paraclinical marker to monitor remyelination in MS, for example to study the effect of therapeutical interventions. At present there is no known imaging marker for remyelination.
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Now that the concept of remyelination is gaining acceptance in MS, there is a need for a paraclinical marker to monitor remyelination in MS, for example to study the effect of therapeutical interventions. At present there is no known imaging marker for remyelination.
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Remyelination in multiple sclerosis
2009Remyelination in multiple sclerosis is in most cases insufficient, leading to irreversible disability. Different and nonexclusive factors account for this repair deficit. Local inhibitors of the differentiation of oligodendrocyte progenitor cells (OPCs) might play a role, as well as axonal factors impairing the wrapping process. Alternatively, a defect
Gabrièle, Piaton +3 more
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Remyelination during optic nerve compression
Journal of the Neurological Sciences, 1980The structural changes produced in cat optic nerve by incremental chronic compression have been investigated by light and electron microscopy. Within the first week extensive demyelination occurred; some fibres underwent Wallerian degeneration and there was marked proliferation of astrocytic processes.
R E, Clifford-Jones +2 more
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