Results 191 to 200 of about 22,271 (244)
Platelets During Myelin Repair in Multiple Sclerosis: Friend or Foe? [PDF]
Rivera FJ +4 more
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A degradable PEGDA-dopamine hydrogel with ROS scavenging capacity supports flexible design for nerve repair. [PDF]
Huang L +6 more
europepmc +1 more source
Adiponectin Fortifies White Matter After Chronic Hypoperfusion-Induced Vascular Dementia. [PDF]
Miao W +14 more
europepmc +1 more source
Therapeutic targeting of oligodendrocytes in an agent-based model of multiple sclerosis. [PDF]
Weatherley GR +3 more
europepmc +1 more source
<i>Aurkb</i> deficiency disrupts microglial development, homeostasis and hinders remyelination following cuprizone-induced demyelination. [PDF]
Yan W +12 more
europepmc +1 more source
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Remyelination in multiple sclerosis
Progress in Brain Research, 2009Remyelination in multiple sclerosis is in most cases insufficient, leading to irreversible disability. Different and nonexclusive factors account for this repair deficit. Local inhibitors of the differentiation of oligodendrocyte progenitor cells (OPCs) might play a role, as well as axonal factors impairing the wrapping process. Alternatively, a defect
Anna Williams +2 more
exaly +3 more sources
REMYELINATION IN MULTIPLE SCLEROSIS
Journal of Neuropathology and Experimental Neurology, 1978AbstractChronic plaques in central nervous system tissue fixed by in situe perfusion for electron microscopy were examined for evidence of remyelination in 2 patients with multiple sclerosis (MS). Fibers with abnormal central myelin sheaths of several types were found at the margins of most of the plaques studied. The most common of these were: (1) the
J W, Prineas, F, Connell
openaire +2 more sources
NeuroReport, 2002
Remyelination of demyelinated axons in the CNS is a regenerative process that, like many others, becomes less efficient with age. This article reviews a series of studies in which toxin models of demyelination have been used to characterize this phenomenon.
Robin J M, Franklin +2 more
openaire +2 more sources
Remyelination of demyelinated axons in the CNS is a regenerative process that, like many others, becomes less efficient with age. This article reviews a series of studies in which toxin models of demyelination have been used to characterize this phenomenon.
Robin J M, Franklin +2 more
openaire +2 more sources

