Results 271 to 280 of about 199,098 (313)
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Pathophysiology of Ischemia-Reperfusion Injury
Transplantation Proceedings, 2006Our studies characterized the intestinal microcirculatory changes in canine models of intestinal hypoperfusion (hemorrhagic shock) or ischemia-reperfusion (small bowel autotransplantation). The villus microcirculatory parameters (functional capillary density, mean red blood cell velocity) were observed by intravital microscopy using orthogonal ...
J, Kaszaki +3 more
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Renal ischaemia-reperfusion injury
British Journal of Surgery, 1996Ischaemia-reperfusion injury is a complex interrelated sequence of events that classically involves the vascular endothelium and activated leucocytes. During the ischaemic phase the endothelium is primed both to produce free radicals and to secrete chemoattractants.
Weight, SC, Bell, PRF, Nicholson, ML
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Struggling With Ischemia Reperfusion Injury
Journal of Craniofacial Surgery, 2007Ischemia reperfusion (I/R) injury is a complicated injury that compels either the patient or the practitioner. In the literature, there still is a debate over how to manage the injury. Typically, no single treatment is feasible. So far, numerous substances have been used as treatments and the majority of these are antioxidants.
Oguz, Kayiran +3 more
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ISCHAEMIA‐REPERFUSION INJURY TO THE INTESTINE
Australian and New Zealand Journal of Surgery, 1998Ischaemia‐reperfusion injury (IRI) is of obvious relevance in situations where there is an interruption of blood supply to the gut, as in vascular surgery, or in the construction of free intestinal grafts. It is now appreciated that IRI also underlies the gut dysfunction that occurs in early shock, sepsis, and trauma.
S E, Kong +4 more
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Mechanisms of myocardial reperfusion injury
The Annals of Thoracic Surgery, 1999Reperfusion of the ischemic myocardium results in irreversible tissue injury and cell necrosis, leading to decreased cardiac performance. While early reperfusion of the heart is essential in preventing further tissue damage due to ischemia, reintroduction of blood flow can expedite the death of vulnerable, but still viable, myocardial tissue, by ...
J L, Park, B R, Lucchesi
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New horizons (Baltimore, Md.), 1996
Reperfusion injury, precipitated by lack of oxygen, is likely to play a major role in many clinical conditions, including shock, coronary artery occlusion disease, and solid organ transplantation. Certain tissues, such as the intestinal mucosa, may be especially susceptible because of the specific microvascular anatomy.
A, Ar'Rajab, I, Dawidson, R, Fabia
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Reperfusion injury, precipitated by lack of oxygen, is likely to play a major role in many clinical conditions, including shock, coronary artery occlusion disease, and solid organ transplantation. Certain tissues, such as the intestinal mucosa, may be especially susceptible because of the specific microvascular anatomy.
A, Ar'Rajab, I, Dawidson, R, Fabia
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Journal of vascular nursing : official publication of the Society for Peripheral Vascular Nursing, 1994
Reperfusion injury occurs after flow is restored to an ischemic area in an effort to salvage limbs and prevent amputation. Skeletal muscle damage occurs during ischemia as well as during reperfusion. Literature review supports the theory that reperfusion of the ischemic area may cause a chain of events that results in irreversible necrosis. Neutrophils,
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Reperfusion injury occurs after flow is restored to an ischemic area in an effort to salvage limbs and prevent amputation. Skeletal muscle damage occurs during ischemia as well as during reperfusion. Literature review supports the theory that reperfusion of the ischemic area may cause a chain of events that results in irreversible necrosis. Neutrophils,
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Neutrophils and myocardial reperfusion injury
Pharmacology & Therapeutics, 1996Ischaemia induces an acute inflammatory response in myocardial tissue with an early phase of neutrophil accumulation, which is accelerated by reperfusion. In experimental models, interventions that deplete neutrophils or inhibit their function cause a significant reduction in myocardial infarct size. These cells, therefore, may exacerbate tissue injury
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