Results 211 to 220 of about 50,772 (264)
Abemaciclib Inhibits Retinoblastoma Tumor Growth by Targeting CDK1/2. [PDF]
Yang H +6 more
europepmc +1 more source
Exploring tear biomarkers with shotgun proteomics for retinoblastoma diagnosis: a pilot study. [PDF]
Rodríguez-Rodríguez A +7 more
europepmc +1 more source
Retinoblastoma and Its Tumor Microenvironment. [PDF]
Vaithianathan AM, Zanazzi G.
europepmc +1 more source
One hotspot <i>RB1</i> mutation disrupt <i>RB1</i> function founded in a Chinese patient. [PDF]
Liu Y +6 more
europepmc +1 more source
PTTG1-mediated the transcription of c-myc promotes retinoblastoma progression. [PDF]
Feng J, Ye Y, Cheng F, Wu Y.
europepmc +1 more source
Amentoflavone Suppresses Stemness of Retinoblastoma Cell via Targeting Smoothened (SMO) Protein. [PDF]
Li C +5 more
europepmc +1 more source
Centromere protein E as a novel biomarker and potential therapeutic target for retinoblastoma
Retinoblastoma is the most common intraocular malignancy during childhood. Currently, there is no effective treatment for metastatic retinoblastoma. We investigated potential biomarkers of retinoblastoma by utilizing three datasets from a public database.
Ke Shi, Xinyue Zhu, Jiali Wu
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The retinoblastoma protein and its relatives
Seminars in Cancer Biology, 1995The retinoblastoma tumor suppressor gene is inactivated in retinoblastomas and a variety of other tumor types. In addition, it is one of several cellular proteins targeted by the transforming proteins of the small DNA tumor viruses. At least two other cellular proteins that are targeted by the viral transforming proteins are structurally and ...
Peter Whyte
exaly +3 more sources
Mutation, absence or abnormal functioning of retinoblastoma protein are fundamental elements of uncontrolled growth in human cancer. In this study, we analyze the expression of retinoblastoma protein and phosphorylated retinoblastoma protein in ...
Alexander Roesch +2 more
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The Family of Retinoblastoma Proteins
Critical Reviews in Eukaryotic Gene Expression, 2001Our understanding of how the retinoblastoma family members, pRB/p105, pRB2/p130, and pRBL1/p107, regulate cellular properties has progressed significantly. Mechanisms have been described regarding how these proteins utilize properties of additional factors, such as histone deacetylases, to negatively regulate transcription.
Stiegler P., Giordano A.
openaire +4 more sources

