Results 161 to 170 of about 99,117 (203)

RhoA/Rho-Kinase Signaling in Vascular Smooth Muscle and Endothelium: Mechanistic Insights and Translational Implications in Hypertension. [PDF]

Biomolecules
Randar S, Silva-Velasco DL, Priviero F, Webb RC.   +3 more
europepmc   +1 more source

Inhibiting the Secreted RGDKGE Collagen Peptide Selectively Controls CD8⁺ T-Cell Migration on Denatured Collagen-IV and Enhances Their Accumulation in Tumors. [PDF]

Am J Pathol
Han X, Caron JM, Brooks PC.
europepmc   +1 more source

MiRNA-140-5p Modulates Cartilage Mechanical Environment by Preserving Surface Stress Homeostasis. [PDF]

Cartilage
Li B, Chen G, Han L, Li H, Zou N.
europepmc   +1 more source

Ubiquitylation-dependent Rap2 activation regulates lamellipodia dynamics during cell migration. [PDF]

J Cell Sci
Neumann A, Sampath R, Mayerhofer E, Mikalayeva V, Skeberdis VA, Sarapinienė I, Prekeris R.   +6 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Regulation of RhoA GTPase and various transcription factors in the RhoA pathway

Journal of Cellular Physiology, 2018
RhoA GTPase plays a variety of functions in regulation of cytoskeletal proteins, cellular morphology, and migration along with various proliferation and transcriptional activity in cells. RhoA activity is regulated by guanine nucleotide exchange factors (GEFs), GTPase activating proteins (GAPs), and the guanine nucleotide dissociation factor (GDI). The
Rokibul Islam, Jae-Bong Park
exaly   +3 more sources

Redox Regulation of RhoA

Biochemistry, 2006
We have previously shown that redox agents including superoxide anion radical and nitrogen dioxide can react with GXXXXGK(S/T)C motif-containing GTPases (i.e., Rac1, Cdc42, and RhoA) to stimulate guanine nucleotide release. We now show that the reaction of RhoA with redox agents leads to different functional consequences from that of Rac1 and Cdc42 due
Jongyun, Heo, Kimberly W, Raines, Viorel, Mocanu, Sharon L, Campbell   +3 more
openaire   +2 more sources